Professor, Pulmonology, University of Colorado Denver School of Medicine
Pulmonology
Dr. Andrew Fontenot, MD
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Andrew Fontenot works in the field of Pulmonology. He researched medicine at University of Alabama Medical Center. University of Alabama Medical Center is ranked 37 in research and 17 in primary care medicine He has 2 awards "CMS Meaningful Use Stage 1 Certification" and "Elected Member". Andrew Fontenot is also published. He has 69 publications published. The lastest publication: "Expansions of T-cell subsets expressing particular T-cell receptor variable regions in chronic beryllium disease.' Andrew Fontenot is registered with Medicare.
Publications
- MyD88 dependence of beryllium-induced dendritic cell trafficking and CD4(+) T-cell priming.
- Lymphocytic Alveolitis is Associated with the Accumulation of Functionally Impaired HIV-specific T Cells in the Lung of ART-naive Subjects.
- Structural basis of Chronic beryllium disease: linking allergic hypersensitivity and autoimmunity.
- Regulatory T cells modulate granulomatous inflammation in an HLA-DP2 transgenic murine model of beryllium-induced Disease.
- Identification of Multiple Public TCR Repertoires in Chronic Beryllium Disease.
- Allergy and hypersensitivity.
- Alterations in the Gut Microbiota Associated with HIV-1 Infection.
- Impaired function of ctla-4 in the lungs of patients with Chronic beryllium disease contributes to persistent inflammation.
- Use of bronchoalveolar lavage to assess the Respiratory microbiome: signal in the noise.
- Widespread Colonization of the Lung by Tropheryma whipplei in HIV Infection.
- In vivo blockade of the PD-1 receptor suppresses HIV-1 viral loads and improves CD4+ T cell levels in humanized mice.
- Influence of MHC class II in susceptibility to beryllium sensitization and Chronic beryllium disease.
- Beryllium-induced tumor necrosis factor-alpha production by CD4+ T cells is mediated by HLA-DP.
- Drug-induced diffuse alveolar hemorrhage syndromes and vasculitis.
- Beryllium-ferritin: lymphocyte proliferation and macrophage apoptosis in Chronic beryllium disease.
- An animal model of autoimmune Emphysema.
- Beryllium-stimulated reactive oxygen species and macrophage apoptosis.
- Activation pathways implicate anti-HLA-DP and anti-LFA-1 antibodies as lead candidates for intervention in Chronic berylliosis.
- Frequency of beryllium-specific, TH1-type cytokine-expressing CD4+ T cells in patients with beryllium-induced Disease.
- Class II major histocompatibility complex-peptide tetramer staining in relation to functional avidity and T cell receptor diversity in the mouse CD4(+) T cell response...
- Oligoclonal CD4+ T cells in the lungs of patients with severe Emphysema.
- Gene expression influences on metal immunomodulation.
- Genetic susceptibility and immune-mediated destruction in beryllium-induced Disease.
- Frequency of beryllium-specific, central memory CD4+ T cells in blood determines proliferative response.
- Beryllium presentation to CD4+ T cells is dependent on a single amino acid residue of the MHC class II beta-chain.
- Functional and phenotypic characterization of CD57+CD4+ T cells and their association with HIV-1-induced T cell dysfunction.
- Self-presentation of beryllium by BAL CD4+ T cells: T cell-T cell interactions and their potential role in Chronic beryllium disease.
- T cell recognition in Chronic beryllium disease.
- Activated oligoclonal CD4+ T cells in the lungs of patients with severe Emphysema.
- Recombinant HLA-DP2 binds beryllium and tolerizes beryllium-specific pathogenic CD4+ T cells.
- Beryllium-induced TNF-alpha production is transcription-dependent in Chronic beryllium disease.
- Regulatory role of gammadelta T cells in the recruitment of CD4+ and CD8+ T cells to lung and subsequent Pulmonary fibrosis.
- Cellular and humoral autoimmunity directed at bile duct epithelia in murine biliary atresia.
- CD57 expression correlates with alveolitis severity in subjects with beryllium-induced Disease.
- Oligoclonal expansions of CD4+ and CD8+ T-cells in the target organ of patients with biliary atresia.
- Programmed death 1 expression on HIV-specific CD4+ T cells is driven by viral replication and associated with T cell dysfunction.
- Up-regulation of programmed death-1 expression on beryllium-specific CD4+ T cells in Chronic beryllium disease.
- gammadelta T cells: an important source of IL-17.
- 4-1BB enhances proliferation of beryllium-specific T cells in the lung of subjects with Chronic beryllium disease.
- Th17-polarized immune response in a murine model of hypersensitivity pneumonitis and lung Fibrosis.
- Metal-induced diffuse lung Disease.
- Decreased 4-1BB expression on HIV-specific CD4+ T cells is associated with sustained viral replication and reduced IL-2 production.
- IL-17A-expressing T cells are essential for bacterial clearance in a murine model of hypersensitivity Pneumonitis.
- CD27 expression on CD4+ T cells differentiates effector from regulatory T cell subsets in the Lung.
- Gammadelta T cells and Th17 cytokines in hypersensitivity pneumonitis and lung Fibrosis.
- Deficient and dysfunctional regulatory T cells in the lungs of Chronic beryllium disease subjects.
- Crystal structure of HLA-DP2 and implications for Chronic beryllium disease.
- Linking genetic susceptibility and T cell activation in beryllium-induced Disease.
- Regulation of virus-specific CD4+ T cell function by multiple costimulatory receptors during Chronic HIV infection.
- ?? T cells protect against lung Fibrosis via IL-22.
- Altered immune phenotype in peripheral blood cells of patients with scleroderma-associated Pulmonary hypertension.
- Immunomodulatory strategies prevent the development of autoimmune Emphysema.
- Defective leukocyte GM-CSF receptor (CD116) expression and function in inflammatory bowel Disease.
- Mutagenesis of beryllium-specific TCRs suggests an unusual binding topology for antigen recognition.
- Beryllium-specific CD4+ T cells in blood as a biomarker of Disease progression.
- Regulatory T cells and lung Fibrosis: a good cell gone bad.
- Beryllium skin patch testing to analyze T cell stimulation and granulomatous inflammation in the Lung.
- Development of neurologic symptoms in a 26-year-old woman following recovery from methanol intoxication.
- Target organ localization of memory CD4(+) T cells in patients with Chronic beryllium disease.
- CD28 costimulation independence of target organ versus circulating memory antigen-specific CD4+ T cells.
- Chronic beryllium disease: immune-mediated destruction with implications for organ-specific autoimmunity.
- Expansions of T-cell subsets expressing particular T-cell receptor variable regions in Chronic beryllium disease.
- Identification of pathogenic T cells in patients with beryllium-induced lung Disease.
- Beryllium presentation to CD4+ T cells underlies disease-susceptibility HLA-DP alleles in Chronic beryllium disease.
- Chronic beryllium disease: T cell recognition of a metal presented by HLA-DP.
- Hypoxemia explained 36 years later.
- Pulmonary opacities and glomerulonephritis in a 15-year-old boy.
- The Impact of Antiretroviral Therapy on Lung Immunology.
Schools
Louisiana State University School Of Medicine In New Orleans
University Of Colorado University Hospital Program
Procedures Preformed
- Bronchoscopy
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asthma
- Atelectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
- Elected Member
Education
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University of Colorado -
University of Alabama Medical Center -
Louisiana State University School of Medicine in New Orleans
Hospital
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University of Colorado Hospital
Drug Facts
| NPI NUMBER | 1962592642 | |
| NPPES Provider LastName | FONTENOT | |
| NPPES Provider FirstName | ANDREW | |
| NPPES Provider ZIPCode | 800452545 | |
| NPPES Provider State | CO | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 90.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1962592642 |
| Last Name Of The Provider | FONTENOT |
| First Name Of The Provider | ANDREW |
| View All |
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