Associate Professor, Pathology, University of Michigan Medical School
Pathology
Dr. Andrew P. Lieberman, MD
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Andrew P. Lieberman, MD works in the field of Pathology. He went to medical school at University of Maryland School of Medicine. University of Maryland School of Medicine is ranked 37 in research and 52 in primary care medicine He over the years received 1 awards: "Elected Member". Andrew Lieberman is a published physician as well. He has 97 publications published. The latest was: Dementia resulting from dural arteriovenous fistulas: the pathologic findings of venous hypertensive encephalopathy. Andrew Lieberman accepts Medicare.
Publications
- Human spinal autografts of olfactory epithelial stem cells recapitulate donor site histology, maintaining proliferative and differentiation capacity many years after t...
- Genetic and pharmacological evidence implicates cathepsins in Niemann-Pick C cerebellar degeneration.
- Post-Mortem evaluation of amyloid-dopamine terminal positron emission tomography dementia classifications.
- Rarity of the Alzheimer Disease-Protective APP A673T Variant in the United States.
- Effects of Multiple Genetic Loci on Age at Onset in Late-Onset Alzheimer Disease: A Genome-Wide Association Study.
- Differential effects of delayed aging on phenotype and striatal pathology in a murine model of huntington disease.
- Targeting Hsp90/Hsp70-Based Protein Quality Control for Treatment of Adult Onset Neurodegenerative Diseases.
- A model in which heat shock protein 90 targets protein-folding clefts: Rationale for a new approach to neuroprotective treatment of protein folding diseases.
- Anti-androgen flutamide protects male mice from androgen-dependent toxicity in three models of spinal bulbar muscular atrophy.
- Peripheral Androgen Receptor Gene Suppression Rescues Disease in Mouse Models of Spinal and Bulbar Muscular Atrophy.
- TMEM106B is a genetic modifier of frontotemporal lobar degeneration with C9orf72 hexanucleotide repeat expansions.
- Funding anatomic pathology research: a retrospective analysis of an intramural funding mechanism.
- Pathogenic Mechanisms and Therapeutic Strategies in Spinobulbar Muscular Atrophy.
- Radiology-pathology case report: isolated extranodal Rosai-Dorfman disease of the skull base.
- Npc1 acting in neurons and glia is essential for the formation and maintenance of CNS myelin.
- C9ORF72 expansion in a family with bipolar disorder.
- Sources of Technical Variability in Quantitative LC-MS Proteomics: Human Brain Tissue Sample Analysis.
- Activation of Hsp70 reduces neurotoxicity by promoting polyglutamine protein degradation.
- Autophagic dysfunction in a lysosomal storage disorder due to impaired proteolysis.
- A polyglutamine expansion disease protein sequesters PTIP to attenuate DNA repair and increase genomic instability.
- Impaired proteolysis underlies autophagic dysfunction in Niemann-Pick type C Disease.
- Pineal region myeloid sarcoma.
- Autophagy in lysosomal storage disorders.
- Ryanodine receptor antagonists adapt NPC1 proteostasis to ameliorate lipid storage in Niemann-Pick type C disease fibroblasts.
- Guidelines for the use and interpretation of assays for monitoring autophagy.
- Genome-wide DNA methylation differences between late-onset Alzheimer's disease and cognitively normal controls in human frontal cortex.
- Macroautophagy Is Regulated by the UPR-Mediator CHOP and Accentuates the Phenotype of SBMA Mice.
- Impaired motoneuronal retrograde transport in two models of SBMA implicates two sites of androgen action.
- Temporal and cell-specific deletion establishes that neuronal Npc1 deficiency is sufficient to mediate neurodegeneration.
- Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's disease.
- Inhibition of hsp70 by methylene blue affects signaling protein function and ubiquitination and modulates polyglutamine protein degradation.
- Common variants at 7p21 are associated with frontotemporal lobar degeneration with TDP-43 inclusions.
- Microarray analysis of gene expression by skeletal muscle of three mouse models of Kennedy disease/spinal bulbar muscular atrophy.
- Small ubiquitin-like modifier (SUMO) modification of the androgen receptor attenuates polyglutamine-mediated aggregation.
- Tau normal function influences Niemann-Pick type C disease pathogenesis in mice and modulates autophagy in NPC1-deficient cells.
- Recovery of function in a myogenic mouse model of spinal bulbar muscular atrophy.
- Overexpression of wild-type androgen receptor in muscle recapitulates polyglutamine disease.
- Lipid trafficking defects increase Beclin-1 and activate autophagy in Niemann-Pick type C disease.
- Genetic variation in C57BL/6 ES cell lines and genetic instability in the Bruce4 C57BL/6 ES cell line.
- Inflammatory dural masses: if it's not one thing, it's another.
- Androgen-dependent pathology demonstrates myopathic contribution to the Kennedy disease phenotype in a mouse knock-in model.
- Abnormalities of germ cell maturation and sertoli cell cytoskeleton in androgen receptor 113 CAG knock-in mice reveal toxic effects of the mutant protein.
- The unfolded protein response modulates toxicity of the expanded glutamine androgen receptor.
- Multiple transcripts of sodium channel SCN8A (Na(V)1.6) with alternative 5'- and 3'-untranslated regions and initial characterization of the SCN8A promoter.
- Castration restores function and neurofilament alterations of aged symptomatic males in a transgenic mouse model of spinal and bulbar muscular atrophy.
- Critical role for DP5/Harakiri, a Bcl-2 homology domain 3-only Bcl-2 family member, in axotomy-induced neuronal cell death.
- Androgen receptor acetylation site mutations cause trafficking defects, misfolding, and aggregation similar to expanded glutamine tracts.
- SUMO, a ubiquitin-like modifier implicated in neurodegeneration.
- Aberrant Autophagic Response in The Muscle of A Knock-in Mouse Model of Spinal and Bulbar Muscular Atrophy.
- Outcomes and complications of fetal tissue transplantation in Parkinson''s disease.
- Interpersonal communication in the context of dementia.
- Long-term experience with selegiline and levodopa in Parkinson's disease.
- Dopamine agonists used as monotherapy in de novo PD patients: comparisons with selegiline.
- Adolf Hitler's cognitive disorder and how it affected his conduct of World War II.
- Safety of intrastriatal neurotransplantation for Huntington's disease patients.
- Outcome following intrastriatal fetal mesencephalic grafts for Parkinson's patients is directly related to the volume of grafted tissue.
- Poly(A) removal is the kinase-regulated step in tumor necrosis factor mRNA decay.
- Refractory giant cell arteritis with spinal cord infarction.
- Intratesticular vasculitis simulating a testicular neoplasm.
- MR of thoracic cord compression caused by epidural extramedullary hematopoiesis in myelodysplastic syndrome.
- The identification of methicillin-resistant Staphylococcus aureus in Osler's nodes and Janeway lesions of acute bacterial endocarditis.
- Muckle-Wells syndrome: case report and review of cutaneous pathology.
- Case of the month: April 1997--a 32 year old man with mental status changes and a severe occipital headache.
- Cloning and genomic organization of the mouse gene slc23a1 encoding a vitamin C transporter.
- Impact of the human genome sequence on neurology and neuroscience.
- Massive symptomatic subependymoma of the lateral ventricles: case report and review of the literature.
- Production of tumor necrosis factor and other cytokines by astrocytes stimulated with lipopolysaccharide or a neurotropic virus.
- Dementia in Parkinson''s disease: a post-mortem study in a population of brain donors.
- Mining the genome for causes and cures of neurological disease.
- Gene-toxin interaction as a putative risk factor for Parkinson''s disease with dementia.
- Triplet repeat expansion in neuromuscular disease.
- Can Alzheimer''s type pathology influence the clinical phenotype of Parkinson''s disease?
- Dementia resulting from dural arteriovenous fistulas: the pathologic findings of venous hypertensive encephalopathy.
- Polyglutamine-containing aggregates in neuronal intranuclear inclusion disease.
- Novel insertional presenilin 1 mutation causing Alzheimer disease with spastic paraparesis.
- Autophagy-mediated dendritic cell activation is essential for innate cytokine production and APC function with respiratory syncytial virus responses.
- Androgens regulate the mammalian homologues of invertebrate sex determination genes tra-2 and fox-1.
- Ataxin 1 and ataxin 3 in neuronal intranuclear inclusion disease.
- Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes.
- A cell culture model for androgen effects in motor neurons.
- Cognitive, neuroimaging, and pathological studies in a patient with Pick's disease.
- Androgen receptor mutation in Kennedy''s disease.
- Altered transcriptional regulation in cells expressing the expanded polyglutamine androgen receptor.
- Huntingtin Interacting Protein 1 mutations lead to abnormal hematopoiesis, spinal defects and cataracts.
- Pharmacologic and genetic inhibition of hsp90-dependent trafficking reduces aggregation and promotes degradation of the expanded glutamine androgen receptor without st...
- Tau deletion exacerbates the phenotype of Niemann-Pick type C mice and implicates autophagy in pathogenesis.
- CHIP deletion reveals functional redundancy of E3 ligases in promoting degradation of both signaling proteins and expanded glutamine proteins.
- The pathogenesis of Niemann-Pick type C disease: a role for autophagy?
- Spinal and bulbar muscular atrophy: a motoneuron or muscle disease?
- The androgen receptor''s CAG/glutamine tract in mouse models of neurological disease and cancer.
- Neuropathological heterogeneity in frontotemporal lobar degeneration with TDP-43 proteinopathy: a quantitative study of 94 cases using principal components analysis.
- Conditional Niemann-Pick C mice demonstrate cell autonomous Purkinje cell neurodegeneration.
- Altered RNA splicing contributes to skeletal muscle pathology in Kennedy disease knock-in mice.
- Nodular bilateral amygdala degeneration in demented individuals.
- Prenatal flutamide enhances survival in a myogenic mouse model of spinal bulbar muscular atrophy.
- Autophagy in Niemann-Pick C disease is dependent upon Beclin-1 and responsive to lipid trafficking defects.
Schools
University of Maryland At Baltimore Professional Schools
Hospital Of The University Of Pennsylvania
Ninds
Doctors Specialties
- Neuropathology
- Pathology
Accepted Insurances
- Choice Plus POS II
- Blue Card PPO
- Blue Cross Blue Shield of Illinois PPO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Cofinity PPO
- Commercial Insurance Company
- Emblem Health
- First Health (Coventry Health Care) PPO
- Health Alliance
- Preferred PPO
- Medicaid
- Medicare MCR
- MultiPlan PPO
- PHCS Network PPO
- Tricare
- Choice Plus POS
- Options PPO
- United Healthcare
Awards
- Elected Member
Education
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University of Pennsylvania Health System -
University of Maryland School of Medicine
Hospital
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University of Michigan Hospitals and Health Centers
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1669554614 |
| Last Name Of The Provider | LIEBERMAN |
| First Name Of The Provider | ANDREW |
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