Associate Professor, Internal Medicine, Yale University School of Medicine
Gastroenterology
Dr. Clara Abraham, MD
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Clara Abraham is an expert in the field of Gastroenterology. She went to University of Chicago Pritzker School of Medicine. University of Chicago Pritzker School of Medicine is ranked 10 in research and 19 in primary care medicine Clara Abraham, MD has written publications. She has 21 publications published with PubMed. The lastest paper is: 'A TPL2 (MAP3K8) disease-risk polymorphism increases TPL2 expression thereby leading to increased pattern recognition receptor-initiated caspase-1 and caspase-8 activat...'
Publications
- A TPL2 (MAP3K8) Disease-risk polymorphism increases TPL2 expression thereby leading to increased pattern recognition receptor-initiated caspase-1 and caspase-8 activat...
- T Cell-Extrinsic CD18 Attenuates Antigen-Dependent CD4+ T Cell Activation In Vivo.
- TAM receptor-dependent regulation of SOCS3 and MAPKs contributes to proinflammatory cytokine downregulation following chronic NOD2 stimulation of human macrophages.
- A TNFSF15 Disease-risk polymorphism increases pattern-recognition receptor-induced signaling through caspase-8-induced IL-1.
- Activation of Pattern Recognition Receptors Upregulates Metallothioneins, Thereby Increasing Intracellular Accumulation of Zinc, Autophagy, and Bacterial Clearance by ...
- The IL18RAP Region Disease Polymorphism Decreases IL-18RAP/IL-18R1/IL-1R1 Expression and Signaling through Innate Receptor-Initiated Pathways.
- Pattern Recognition Receptor Signaling in Human Dendritic Cells is Enhanced by ICOS Ligand and Modulated by the Crohn's Disease ICOSLG Risk Allele.
- NOD2 Regulates CXCR3-Dependent CD8+ T Cell Accumulation in Intestinal Tissues with Acute Injury.
- NF-κB1 Inhibits NOD2-Induced Cytokine Secretion through ATF3-Dependent Mechanisms.
- TNFR2 Activates MLCK-Dependent Tight Junction Dysregulation to Cause Apoptosis-Mediated Barrier Loss and Experimental Colitis.
- The influence of depression on quality of life in patients with inflammatory bowel Disease.
- Extended haplotype association study in Crohn's Disease identifies a novel, Ashkenazi Jewish-specific missense mutation in the NF-?B pathway gene, HEATR3.
- Age and CD161 Expression Contribute to Inter-Individual Variation in Interleukin-23 Response in CD8+ Memory Human T Cells.
- NLRP1 and NLRP3 inflammasomes are essential for distinct outcomes of decreased cytokines but enhanced bacterial killing upon chronic Nod2 stimulation.
- Negative regulation of human mononuclear phagocyte function.
- Nod2-Induced Autocrine Interleukin-1 Alters Signaling by ERK and p38 to Differentially Regulate Secretion of Inflammatory Cytokines.
- IL-22BP is regulated by the inflammasome and modulates tumorigenesis in the intestine.
- Effector CD4+ T Cell Expression Signatures and Immune-Mediated Disease Associated Genes.
- IRF5 risk polymorphisms contribute to interindividual variance in pattern recognition receptor-mediated cytokine secretion in human monocyte-derived cells.
- Targeted epithelial tight junction dysfunction causes immune activation and contributes to development of experimental Colitis.
Schools
U Of Chgo Div Of Bio Sci Pritzker Sch Of Med
Conditions Treated
- Abdominal Pain
- Anemia
- Cirrhosis
- Crohn's Disease (Regional Enteritis)
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Doctors Specialties
- Gastroenterology
Accepted Insurances
Education
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University of Chicago -
Rush University Medical Center -
University of Chicago Pritzker School of Medicine
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