Professor, Molecular Medicine, Case Western Reserve University School of Medicine
Gastroenterology
Dr. Claudio Fiocchi, MD
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Doctor Claudio Fiocchi, MD works in the field of Gastroenterology. Claudio Fiocchi, MD is also published. He has 98 publications published. The lastest publication: "Letter: volatile metabolomics of exhaled breath or faecal gas? - authors' reply.'
Publications
- Letter: volatile metabolomics of exhaled breath or faecal gas? - authors' reply.
- CAP-D3 Promotes Bacterial Clearance in Human Intestinal Epithelial Cells by Repressing Expression of Amino Acid Transporters.
- Anti-fibrogenic effects of the anti-microbial peptide cathelicidin in murine colitis-associated fibrosis.
- Review article: breath analysis in inflammatory bowel diseases.
- The Neurotensin-HIF-1α-VEGFα Axis Orchestrates Hypoxia, Colonic Inflammation, and Intestinal Angiogenesis.
- Impact of Abdominal Visceral Adipose Tissue on Disease Outcome in Pediatric Crohn's Disease.
- Fibrosis in Ulcerative Colitis: Mechanisms, Features, and Consequences of a Neglected Problem.
- IL-13Rα2-bearing, type II NKT cells reactive to sulfatide self-antigen populate the mucosa of ulcerative colitis.
- Gene-gene and gene-environment interactions in ulcerative colitis.
- Inflammatory bowel disease: evolutionary concepts in biology, epidemiology, mechanisms and therapy.
- A Novel Approach to Detect Cumulative Genetic Effects and Genetic Interactions in Crohn's Disease.
- Pro-Angiogenic Activity of TLRs and NLRs: A Novel Link Between Gut Microbiota and Intestinal Angiogenesis.
- Genes and 'in-vironment': how will our concepts on the pathophysiology of inflammatory bowel disease develop in the future?
- Towards a 'Cure' for IBD.
- IBD: advances in pathogenesis, complications, diagnosis, and therapy.
- Increased Levels of Survivin, via Association With Heat Shock Protein 90, in Mucosal T Cells From Patients With Crohn's Disease.
- Early versus late immune mediated inflammatory diseases.
- Inflammation-induced endothelial-to-mesenchymal transition: a novel mechanism of intestinal fibrosis.
- Early and late inflammatory bowel disease: why and how are they different?
- Themes in fibrosis and gastrointestinal inflammation.
- Pathogenesis of postoperative recurrence in Crohn''s disease.
- Targeting the innate immune system in pediatric inflammatory bowel disease.
- Probiotic sonicates selectively induce mucosal immune cells apoptosis through ceramide generation via neutral sphingomyelinase.
- News from the "5th International Meeting on Inflammatory Bowel Diseases" CAPRI 2010.
- First international summit on fibrosis in intestinal inflammation: mechanisms and biological therapies.
- Inflammatory bowel disease: Established and evolving considerations on its etiopathogenesis and therapy.
- HCl-induced inflammatory mediators in esophageal mucosa increase migration and production of H2O2 by peripheral blood leukocytes.
- Current perspectives in inflammatory bowel disease: stress response and autophagy, host-microbe mutualism, immune duality and plasticity, and early versus late disease.
- Corticotropin-releasing hormone family of peptides regulates intestinal angiogenesis.
- Epithelial-derived IL-33 and its receptor ST2 are dysregulated in ulcerative colitis and in experimental Th1/Th2 driven enteritis.
- The angiogenic effect of probiotic Bacillus polyfermenticus on human intestinal microvascular endothelial cells is mediated by IL-8.
- Susceptibility genes and overall pathogenesis of inflammatory bowel disease: where do we stand?
- Gene-gene interactions in inflammatory bowel disease: biological and clinical implications.
- Platelet-derived hyaluronidase 2 cleaves hyaluronan into fragments that trigger monocyte-mediated production of proinflammatory cytokines.
- Intestinal fibrosis in IBD--a dynamic, multifactorial process.
- VEGF-A links angiogenesis and inflammation in inflammatory bowel disease pathogenesis.
- Intestinal fibrosis in inflammatory bowel disease - Current knowledge and future perspectives.
- Future of IBD pathogenesis: how much work is left to do?
- What is "physiological" intestinal inflammation and how does it differ from "pathological" inflammation?
- Intestinal fibrosis in inflammatory bowel disease: progress in basic and clinical science.
- Mucosal T cell proliferation and apoptosis in inflammatory bowel disease.
- Hyaluronan (HA) deposition precedes and promotes leukocyte recruitment in intestinal inflammation.
- Inflammatory bowel disease: progress and current concepts of etiopathogenesis.
- Late-breaking news from the "4th International Meeting on Inflammatory Bowel Diseases" Capri, 2006.
- Progress in basic inflammatory bowel disease research.
- Angiogenesis blockade as a new therapeutic approach to experimental colitis.
- Immune regulation by microvascular endothelial cells: directing innate and adaptive immunity, coagulation, and inflammation.
- Enhanced recruitment of CX3CR1+ T cells by mucosal endothelial cell-derived fractalkine in inflammatory bowel disease.
- Gastroesophageal reflux disease-associated esophagitis induces endogenous cytokine production leading to motor abnormalities.
- Management consensus of inflammatory bowel disease for the Asia-Pacific region.
- Probiotics in inflammatory bowel disease: yet another mechanism of action?
- Eosinophilic esophagitis: new insights into an emerging disease.
- Acid-induced release of platelet-activating factor by human esophageal mucosa induces inflammatory mediators in circular smooth muscle.
- Challenges in pediatric inflammatory bowel disease.
- Etiopathogenesis of inflammatory bowel diseases.
- Non-stop progress in inflammatory bowel disease: new players, new understanding, new therapies.
- HCl-induced inflammatory mediators in cat esophageal mucosa and inflammatory mediators in esophageal circular muscle in an in vitro model of esophagitis.
- Angiogenesis as a novel component of inflammatory bowel disease pathogenesis.
- TNF-alpha blockade down-regulates the CD40/CD40L pathway in the mucosal microcirculation: a novel anti-inflammatory mechanism of infliximab in Crohn''s disease.
- Alterations of mesenchymal and endothelial cells in inflammatory bowel diseases.
- Production of IL-1beta, hydrogen peroxide, and nitric oxide by colonic mucosa decreases sigmoid smooth muscle contractility in ulcerative colitis.
- One commensal bacterial molecule--all we need for health?
- In vitro model of acute esophagitis in the cat.
- Hydrogen peroxide reduces lower esophageal sphincter tone in human esophagitis.
- Platelet-activating factor and prostaglandin E2 impair esophageal ACh release in experimental esophagitis.
- Redox equilibrium in mucosal T cells tunes the intestinal TCR signaling threshold.
- Intestinal fibroblast-derived IL-10 increases survival of mucosal T cells by inhibiting growth factor deprivation- and Fas-mediated apoptosis.
- The emergence of inflammatory bowel disease in the Asian Pacific region.
- Platelet activation and the CD40/CD40 ligand pathway: mechanisms and implications for human disease.
- Introduction: First International Symposium on: pediatric inflammatory bowel disease: September 16-18, 2003; Irvington, Virginia, U.S.A.
- An oral supplement enriched with fish oil, soluble fiber, and antioxidants for corticosteroid sparing in ulcerative colitis: a randomized, controlled trial.
- Significance of glucocorticoid receptor expression in colonic mucosal cells of patients with ulcerative colitis.
- Signal transducers and activators of transcription 3 signaling pathway: an essential mediator of inflammatory bowel disease and other forms of intestinal inflammation.
- Inflammatory bowel disease pathogenesis: therapeutic implications.
- Proinflammatory cytokines alter/reduce esophageal circular muscle contraction in experimental cat esophagitis.
- Inflammatory bowel disease: autoimmune or immune-mediated pathogenesis?
- Interleukin 1beta-induced production of H2O2 contributes to reduced sigmoid colonic circular smooth muscle contractility in ulcerative colitis.
- Dual function of the extracellular matrix: stimulatory for cell cycle progression of naive T cells and antiapoptotic for tissue-derived memory T cells.
- Inflammatory bowel disease: the role of environmental factors.
- Inflammatory bowel disease: new insights into mechanisms of inflammation and increasingly customized approaches to diagnosis and therapy.
- Hydrogen peroxide contributes to motor dysfunction in ulcerative colitis.
- Paradoxical decrease of mitochondrial DNA deletions in epithelial cells of active ulcerative colitis patients.
- Platelets in inflammatory bowel disease: clinical, pathogenic, and therapeutic implications.
- Closing fistulas in Crohn''s disease--should the accent be on maintenance or safety?
- Cutting edge: T cells trigger CD40-dependent platelet activation and granular RANTES release: a novel pathway for immune response amplification.
- CD40-mediated immune-nonimmune cell interactions induce mucosal fibroblast chemokines leading to T-cell transmigration.
- A murine model of chronic inflammation-induced intestinal fibrosis down-regulated by antisense NF-kappa B.
- Inflammatory bowel disease-associated gene expression in intestinal epithelial cells by differential cDNA screening and mRNA display.
- More answers and more questions in inflammatory bowel disease.
- Platelets trigger a CD40-dependent inflammatory response in the microvasculature of inflammatory bowel disease patients.
- Atherosclerosis and inflammatory bowel disease: sharing a common pathogenic pathway?
- Inflammatory bowel disease: genetics and much more.
- p53 negatively regulates intestinal immunity by delaying mucosal T cell cycling.
- Critical role of caspases in the regulation of apoptosis and proliferation of mucosal T cells.
- Etiopathogenesis of inflammatory bowel disease: the importance of the pediatric perspective.
- Considerations on a case of "high renal ectopia".
- On the chronic occupational osteopathy of the hands of milkers.
Schools
Faculdade De Ciencias Medicas Da Santa Casa De Sao Paulo
Union Memorial Hospital
Cleveland Clinic
Hospital Da Santa Casa
Doctors Specialties
- Internal Medicine
- Gastroenterology
Accepted Insurances
Education
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Cleveland Clinic Foundation -
Faculty Of Medical Sciences of Santa Casa De Sao Paulo
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