Professor, Neurology, UCLA School of Medicine
Neurology
Dr. Daniel H. Geschwind, MD
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Daniel Geschwind works in the field of Neurology. He attended Yale University School of Medicine. Yale University School of Medicine is one of the top medical schools in the US. Yale University School of Medicine is ranked 7/57 in Research/PrimaryCare. He received awards:"Elected Member" and "Super Doctor". Daniel Geschwind is a published doctor as well. He has 103 publications published. The latest was: Schizophrenia genetics complements its mechanistic understanding. Dr. Daniel H. Geschwind is registered with Medicare and accepts Medicare payments.
Publications
- A Systems-Level Analysis of the Peripheral Nerve Intrinsic Axonal Growth Program.
- Correspondence between Resting-State Activity and Brain Gene Expression.
- Endocannabinoid signaling mediates oxytocin-driven social reward.
- Gene hunting in autism spectrum disorder: on the path to precision medicine.
- Developmental disorders.
- Social responsiveness, an autism endophenotype: genomewide significant linkage to two regions on chromosome 8.
- Genome engineering of isogenic human ES cells to model autism disorders.
- A Multiancestral Genome-Wide Exome Array Study of Alzheimer Disease, Frontotemporal Dementia, and Progressive Supranuclear Palsy.
- Leveraging genetics and genomics to define the causes of mental illness.
- Exogenous and evoked oxytocin restores social behavior in the Cntnap2 mouse model of autism.
- Rarity of the Alzheimer Disease-Protective APP A673T Variant in the United States.
- Effects of Multiple Genetic Loci on Age at Onset in Late-Onset Alzheimer Disease: A Genome-Wide Association Study.
- Altered network connectivity in frontotemporal dementia with C9orf72 hexanucleotide repeat expansion.
- Increased CYFIP1 dosage alters cellular and dendritic morphology and dysregulates mTOR.
- Exome sequencing in the clinical diagnosis of sporadic or familial cerebellar ataxia.
- A Genome-wide Association Study of Autism Using the Simons Simplex Collection: Does Reducing Phenotypic Heterogeneity in Autism Increase Genetic Homogeneity?
- A quantitative framework to evaluate modeling of cortical development by neural stem cells.
- Modest Impact on Risk for Autism Spectrum Disorder of Rare Copy Number Variants at 15q11.2, Specifically Breakpoints 1 to 2.
- Convergence of genes and cellular pathways dysregulated in autism spectrum disorders.
- The neurogenetics of atypical parkinsonian disorders.
- Mutation of Senataxin Alters Disease-Specific Transcriptional Networks in Patients with Ataxia with Oculomotor Apraxia Type 2.
- Transcriptional landscape of the prenatal human brain.
- Genes for endosomal NHE6 and NHE9 are misregulated in autism brains.
- An epigenetic signature in peripheral blood associated with the haplotype on 17q21.31, a risk factor for neurodegenerative tauopathy.
- Disentangling the heterogeneity of autism spectrum disorder through genetic findings.
- Novel roles for osteopontin and clusterin in peripheral motor and sensory axon regeneration.
- Integrative functional genomic analyses implicate specific molecular pathways and circuits in autism.
- Using large clinical data sets to infer pathogenicity for rare copy number variants in autism cohorts.
- Cortical evolution: judge the brain by its cover.
- Intellectual Disability Is Associated with Increased Runs of Homozygosity in Simplex Autism.
- Adjusting Head Circumference for Covariates in Autism: Clinical Correlates of a Highly Heritable Continuous Trait.
- Sex differences in autism spectrum disorders.
- Population structure confounds autism genetic classifier.
- Sumoylated MEF2A coordinately eliminates orphan presynaptic sites and promotes maturation of presynaptic boutons.
- Neurodegenerative Disease Phenotypes in Carriers of MAPT p.A152T, A Risk Factor for Frontotemporal Dementia Spectrum Disorders and Alzheimer Disease.
- TDP-43 frontotemporal lobar degeneration and autoimmune Disease.
- Mutations in SLC20A2 are a major cause of familial idiopathic basal ganglia calcification.
- QTL replication and targeted association highlight the nerve growth factor gene for nonverbal communication deficits in autism spectrum disorders.
- The Disruption of Celf6, a Gene Identified by Translational Profiling of Serotonergic Neurons, Results in Autism-Related Behaviors.
- Autism recurrence in half siblings: strong support for genetic mechanisms of transmission in ASD.
- Rare inherited variation in autism: beginning to see the forest and a few trees.
- Retooling spare parts: gene duplication and cognition.
- Mutations in PDYN are not responsible for multiple system atrophy.
- C9ORF72 expansion is not a significant cause of sporadic spinocerebellar ataxia.
- RBFOX1 regulates both splicing and transcriptional networks in human neuronal development.
- Human-specific transcriptional networks in the brain.
- Frontotemporal dementia due to C9ORF72 mutations: Clinical and imaging features.
- Familial cortical myoclonus with a mutation in NOL3.
- Autism genetics: searching for specificity and convergence.
- Memory performance and fMRI signal in presymptomatic familial Alzheimer's Disease.
- Genome-wide Transcriptome Profiling Reveals the Functional Impact of Rare De Novo and Recurrent CNVs in Autism Spectrum Disorders.
- Network organization of the huntingtin proteomic interactome in Mammalian brain.
- Genomic medicine enters the neurology clinic.
- Evidence for a role of the rare p.A152T variant in MAPT in increasing the risk for FTD-spectrum and Alzheimer's Diseases.
- Differential transcriptional profiles mediated by exposure to the cannabinoids cannabidiol and Δ9-tetrahydrocannabinol in BV-2 microglial cells.
- Proteomic changes in cerebrospinal fluid of presymptomatic and affected persons carrying familial Alzheimer Disease mutations.
- DISC1: A Schizophrenia Gene with Multiple Personalities.
- A gene expression phenotype in lymphocytes from Friedreich ataxia patients.
- A novel approach of homozygous haplotype sharing identifies candidate genes in autism spectrum disorder.
- Genome-wide analysis of a wnt1-regulated transcriptional network implicates neurodegenerative pathways.
- Absence of CNTNAP2 leads to epilepsy, neuronal migration abnormalities, and core autism-related deficits.
- Cortical and Hippocampal Atrophy in Patients with Autosomal Dominant Familial Alzheimer's Disease.
- Functional genomic analyses identify pathways dysregulated by progranulin deficiency, implicating wnt signaling.
- Expanded GGGGCC Hexanucleotide Repeat in Noncoding Region of C9ORF72 Causes Chromosome 9p-Linked FTD and ALS.
- Regulation of MET by FOXP2, genes implicated in higher cognitive dysfunction and autism risk.
- Strategies for aggregating gene expression data: The collapseRows R function.
- Foxp2 regulates gene networks implicated in neurite outgrowth in the developing brain.
- The Human Brain in a Dish: The Promise of iPSC-Derived Neurons.
- Gene expression profiling of R6/2 transgenic mice with different CAG repeat lengths reveals genes associated with Disease onset and progression in Huntington's Disease.
- Transcriptomic analysis of autistic brain reveals convergent molecular pathology.
- Suberoylanilide Hydroxamic Acid (Vorinostat) Up-regulates Progranulin Transcription: RATIONAL THERAPEUTIC APPROACH TO FRONTOTEMPORAL DEMENTIA.
- Mithramycin Is a Gene-Selective Sp1 Inhibitor That Identifies a Biological Intersection between Cancer and Neurodegeneration.
- Neurons show distinctive DNA methylation profile and higher interindividual variations compared with non-neurons.
- Common variants at MS4A4/MS4A6E, CD2AP, CD33 and EPHA1 are associated with late-onset Alzheimer's Disease.
- Effects of risk genes on BOLD activation in presymptomatic carriers of familial Alzheimer's Disease mutations during a novelty encoding task.
- Candidate Screening of the TRPC3 Gene in Cerebellar Ataxia.
- Specific functions for ERK/MAPK signaling during PNS development.
- A systems level, functional genomics analysis of chronic epilepsy.
- Using iPSC-derived neurons to uncover cellular phenotypes associated with Timothy Syndrome.
- An age-related sprouting transcriptome provides molecular control of axonal sprouting after stroke.
- Increased fMRI signal with age in familial Alzheimer's Disease mutation carriers.
- Altered functional connectivity in frontal lobe circuits is associated with variation in the autism risk gene CNTNAP2.
- Human brain evolution: harnessing the genomics (r)evolution to link genes, cognition, and behavior.
- Detecting network modules in fMRI time series: a weighted network analysis approach.
- Divergence of human and mouse brain transcriptome highlights Alzheimer Disease pathways.
- Language-related Cntnap2 gene is differentially expressed in sexually dimorphic song nuclei essential for vocal learning in songbirds.
- Brief report: Under-representation of African americans in autism genetic research: a rationale for inclusion of subjects representing diverse family structures.
- Connecting genes to brain in the autism spectrum disorders.
- Signaling to transcription networks in the neuronal retrograde injury response.
- Identification of differentially expressed proteins in murine embryonic and postnatal cortical neural progenitors.
- Disruption of astrocyte STAT3 signaling decreases mitochondrial function and increases oxidative stress in vitro.
- Autism: the ups and downs of neuroligin.
- Genome-wide analyses of exonic copy number variants in a family-based study point to novel autism susceptibility genes.
- Functional and evolutionary insights into human brain development through global transcriptome analysis.
- Endogenous Wnt signaling maintains neural progenitor cell potency.
- Kinetic profile of the transcriptome changes induced in the choroid plexus by peripheral inflammation.
- Association and mutation analyses of 16p11.2 autism candidate genes.
- Cell lineage and regional identity of cultured spinal cord neural stem cells and comparison to brain-derived neural stem cells.
- Wnt-pathway activation during the early stage of neurodegeneration in FTDP-17 mice.
- Advances in autism.
- The Genetics-Driven Revival in Neuropsychiatric Drug Development.
- Schizophrenia genetics complements its mechanistic understanding.
Schools
Yale University
UCLA Med Center
UCLA Med CtrUCLA D Geffen School
Conditions Treated
- Alzheimer's Disease
- Cerebral Artery Thrombosis
- Gait Abnormality
- Insomnia
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Doctors Specialties
- Neurology
Accepted Insurances
- Aetna HMO
- Choice Plus POS II
- Blue Card PPO
- CaliforniaCare HMO
- Advantage Preferred DirectAccess Plus PPO
- Blue Cross Blue Shield of California PPO
- Pathway X Direct Access PPO
- Pathway X Tiered Direct Access
- Select Plus HMO
- Select PPO
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- First Health (Coventry Health Care) PPO
- Blue & Gold HMO
- Covered California - Bronze PPO
- Employer Group HMO
- Health Net PPO
- Individual and Family PPO
- Kaiser PHCS Network PPO
- MultiPlan PPO
- PHCS Network PPO
- Heritage Signature
- Choice Plus POS
- Navigate HMO
- Navigate POS
- Options PPO
- PacifiCare HMO
Awards
- Elected Member
- Super Doctor
Education
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UCLA Medical Center -
Yale University School of Medicine
Hospital
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Ronald Reagan UCLA Medical Center
Drug Facts
| NPI NUMBER | 1730102310 | |
| NPPES Provider LastName | GESCHWIND | |
| NPPES Provider FirstName | DANIEL | |
| NPPES Provider ZIPCode | 900950001 | |
| NPPES Provider State | CA | |
| Specialty Description | Neurology | |
| Total Claim Count | 36.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1730102310 |
| Last Name Of The Provider | GESCHWIND |
| First Name Of The Provider | DANIEL |
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