Professor
Cardiology
Dr. Daniel T. Eitzman, MD
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Daniel Eitzman practices Cardiology. He graduated from Wayne State University School of Medicine. Wayne State University School of Medicine is ranked 63 in research and 85 in primary care medicine He over the years received 1 awards: "Elected Member". Daniel T. Eitzman is a published doctor as well. He has 39 publications published. The latest was: Renal denervation attenuates progression of atherosclerosis in apolipoprotein e-deficient mice independent of blood pressure lowering. He is registered with Medicare.
Publications
- Renal denervation attenuates progression of atherosclerosis in apolipoprotein e-deficient mice independent of blood pressure lowering.
- Imaging body fat: techniques and cardiometabolic implications.
- Perivascular Adipose Tissue in Vascular Function and Disease: A Review of Current Research and Animal Models.
- Increased stroke size following MCA occlusion in a mouse model of sickle cell Disease.
- Peptidylarginine Deiminase Inhibition Reduces Vascular Damage and Modulates Innate Immune Responses in Murine Models of Atherosclerosis.
- Leptin-induced endothelial dysfunction is mediated by sympathetic nervous system activity.
- Leptin in Thrombosis and Atherosclerosis.
- Acute myocardial infarction leads to acceleration of atherosclerosis.
- Paradoxical protection from atherosclerosis and thrombosis in a mouse model of sickle cell Disease.
- ?(1,3)-Fucosyltransferases FUT4 and FUT7 Control Murine Susceptibility to Thrombosis.
- Paradoxical Roles of Perivascular Adipose Tissue in Atherosclerosis and Hypertension.
- P-selectin glycoprotein ligand-1 inhibition blocks increased leukocyte-endothelial interactions associated with sickle cell Disease in mice.
- Haploinsufficiency of E-selectin ligand-1 is associated with reduced atherosclerotic plaque macrophage content while complete deficiency leads to early embryonic letha...
- Type I interferons modulate vascular function, repair, thrombosis, and plaque progression in murine models of lupus and atherosclerosis.
- Obesity-Induced Endothelial Dysfunction Is Prevented by Deficiency of P-selectin Glycoprotein Ligand-1.
- P-selectin glycoprotein ligand-1 deficiency leads to cytokine resistance and protection against atherosclerosis in apolipoprotein E deficient mice.
- P-selectin glycoprotein ligand-1 regulates adhesive properties of the endothelium and leukocyte trafficking into adipose tissue.
- Monocyte chemoattractant protein-1 deficiency protects against visceral fat-induced atherosclerosis.
- Animal models of thrombosis.
- Visceral adipose tissue inflammation accelerates atherosclerosis in apolipoprotein E-deficient mice.
- Murine models of vascular thrombosis (Eitzman series).
- Valsartan Improves Insulin Sensitivity without Altering Vascular Function in Healthy Overweight Adults without the Metabolic Syndrome.
- Blood pressure and vascular effects of leptin in humans.
- Alpha-galactosidase A in vascular Disease.
- Leptin regulates neointima formation after arterial injury through mechanisms independent of blood pressure and the leptin receptor/STAT3 signaling pathways involved i...
- Alpha-galactosidase A deficiency leads to increased tissue fibrin deposition and thrombosis in mice homozygous for the factor V Leiden mutation.
- Atherosclerosis in mice is not affected by a reduction in tissue factor expression.
- Adiponectin: vascular protection from the fat?
- Recombinant leptin promotes atherosclerosis and thrombosis in apolipoprotein E-deficient mice.
- Homozygosity for factor V Leiden leads to enhanced thrombosis and atherosclerosis in mice.
- Alpha-galactosidase A deficiency accelerates atherosclerosis in mice with apolipoprotein E deficiency.
- Ventricular tachycardia associated with transMyocardial migration of an epicardial pacing wire.
- Fabry Disease in mice is associated with age-dependent susceptibility to vascular thrombosis.
- Lethal perinatal thrombosis in mice resulting from the interaction of tissue factor pathway inhibitor deficiency and factor V Leiden.
- Effect of leptin on arterial thrombosis following vascular injury in mice.
- Competitive bidding for interventional cardiology supplies: lessons learned during round 2.
- Heparin cofactor II inhibits arterial thrombosis after endothelial injury.
- Mebendazole reduces vascular smooth muscle cell proliferation and neointimal formation following vascular injury in mice.
Schools
Wayne State University
University Of Mi Hosps and Health Ctrs
Procedures Preformed
- Cardiac Catheterization (incl. Coronary Angiography)
- Coronary Angioplasty, Atherectomy and Stent
Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Aneurysm
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
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Doctors Specialties
- Cardiology
- Interventional Cardiology
Accepted Insurances
Awards
- Elected Member
Education
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University of Michigan Hospitals and Health Centers -
Wayne State University School of Medicine
Hospital
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University of Michigan Hospitals and Health Centers
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1073698809 |
| Last Name Of The Provider | EITZMAN |
| First Name Of The Provider | DANIEL |
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