Physician
Infectious Disease
Dr. David J. Kusner, MD
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David Kusner is an expert in the field of Infectious Disease. He researched medicine at Harvard Medical School. Harvard Medical School is one of the top medical schools in the US. Harvard Medical School is ranked 1 in research and 12 in primary care medicine He received 1 awards: "Elected Member". David J. Kusner is a published physician. He has 34 papers published. The most current publication "Fc gamma receptor-mediated activation of phospholipase D regulates macrophage phagocytosis of IgG-opsonized particles." David Kusner accepts Medicare payments and is listed with Medicare.gov.
Publications
- Paracrine WNT5A signaling inhibits expansion of tumor-initiating cells.
- ROR1, an embryonic protein with an emerging role in cancer biology.
- Coordinate regulation of sphingosine kinase and actin dynamics.
- MHC Class II levels and intracellular localization in human dendritic cells are regulated by calmodulin kinase II.
- The localization and activity of sphingosine kinase 1 are coordinately regulated with actin cytoskeletal dynamics in macrophages.
- Phospholipase D1 regulates phagocyte adhesion.
- Assay of phospholipase D activity in cell-free systems.
- Calmodulin kinase II regulates the maturation and antigen presentation of human dendritic cells.
- Sphingosine kinase 1 (SK1) is recruited to nascent phagosomes in human macrophages: inhibition of SK1 translocation by Mycobacterium tuberculosis.
- Mechanisms of mycobacterial persistence in tuberculosis.
- Phospholipases D1 and D2 coordinately regulate macrophage phagocytosis.
- Evolutionary conservation of physical and functional interactions between phospholipase D and actin.
- Cutting edge: Mycobacterium tuberculosis blocks Ca2+ signaling and phagosome maturation in human macrophages via specific inhibition of sphingosine kinase.
- Regulation of phospholipase D activity by actin. Actin exerts bidirectional modulation of Mammalian phospholipase D activity in a polymerization-dependent, isoform-spe...
- The regulation of dendritic cell function by calcium-signaling and its inhibition by microbial pathogens.
- Protease priming of neutrophil superoxide production. Effects on membrane lipid order and lateral mobility.
- Cytokine- and LPS-induced synthesis of interleukin-8 from human mesangial cells.
- Syphilis--a reversible cause of nephrotic syndrome in HIV infection.
- Protease-modulation of neutrophil superoxide response.
- Guanosine 5''-[gamma-thio]triphosphate induces membrane localization of cytosol-independent phospholipase D activity in a cell-free system from U937 promonocytic leuco...
- Mycobacterium tuberculosis and its purified protein derivative activate expression of the human immunodeficiency virus.
- Cerebral paragonimiasis.
- ATP-induced potentiation of G-protein-dependent phospholipase D activity in a cell-free system from U937 promonocytic leukocytes.
- Phospholipase D activity in phagocytic leucocytes is synergistically regulated by G-protein- and tyrosine kinase-based mechanisms.
- Activation of phospholipase D is tightly coupled to the phagocytosis of Mycobacterium tuberculosis or opsonized zymosan by human macrophages.
- Association of phospholipase D activity with the detergent-insoluble cytoskeleton of U937 promonocytic leukocytes.
- Fc gamma receptor-mediated activation of phospholipase D regulates macrophage phagocytosis of IgG-opsonized particles.
- Identification of sex-specific differences in surfactant synthesis in rat lung.
- ATP-induced killing of virulent Mycobacterium tuberculosis within human macrophages requires phospholipase D.
- Inhibition of Ca(2+) signaling by Mycobacterium tuberculosis is associated with reduced phagosome-lysosome fusion and increased survival within human macrophages.
- In vitro and in vivo macrophage function can occur independently of SLP-76.
- Mycobacterium tuberculosis phagosomes exhibit altered calmodulin-dependent signal transduction: contribution to inhibition of phagosome-lysosome fusion and intracellul...
- ATP stimulates human macrophages to kill intracellular virulent Mycobacterium tuberculosis via calcium-dependent phagosome-lysosome fusion.
Schools
Harvard Medical School
University Hospital Of Cleveland
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Awards
- Elected Member
Education
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Case Western Reserve University - University Hospitals -
Harvard Medical School
Drug Facts
| NPI NUMBER | 1871562736 | |
| NPPES Provider LastName | KUSNER | |
| NPPES Provider FirstName | DAVID | |
| NPPES Provider ZIPCode | 522452477 | |
| NPPES Provider State | IA | |
| Specialty Description | Infectious Disease | |
| Total Claim Count | 554.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1871562736 |
| Last Name Of The Provider | KUSNER |
| First Name Of The Provider | DAVID |
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