Jeffrey R. Pine Chaired Professor of Medicine and Director, Division of Pulmonary, Allergy, Critical Care and Sleep Medicine, Emory University School of Medicine
Internal Medicine
Dr. David M. Guidot, MD
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David M. Guidot, MD works in the field of Internal Medicine. He went to University of Michigan Medical School. He received 12 awards: "Recipient of the Silver Pear Mentoring Award from the Department of Medicine", "Fellow", "Recipient of the Golden Apple Award for Teaching Excellence by the Internal Medicine housestaff", "Named one of the Best Doctors in America", "Pulmonary Star Award as the outstanding academic faculty member in the Division of Pulmonary, Allergy, & critical Care Medicine", "Department of Medicine Housestaff Teaching Award", "Young Faculty Research Award, Pulmonary Medicine, Southern Section", "Young Investigator Award, Western Section", "Edward Livingston Trudeau Scholar", "Trainee Travel Award", "Research Award" and "Research Fellowship". Doctor David M. Guidot, MD is also published. He has 88 publications published. The lastest publication: "Endotoxin pretreatment in vivo increases the mitochondrial respiratory capacity in rat hepatocytes.' David Guidot accepts Medicare payments and is registered with Medicare.gov.
Publications
- TGFβ1 Mediates Alcohol-Induced Nrf2 Suppression in Lung Fibroblasts.
- Donor alcohol abuse and Lung injury following allograft transplantation: a cautionary tale but also a call to action.
- The Role of Donor Chronic Alcohol Abuse in the Development of Primary Graft Dysfunction in Lung Transplant Recipients.
- Healthy HIV-1-Infected Individuals on Highly Active Antiretroviral Therapy Harbor HIV-1 in Their Alveolar Macrophages.
- Noninfectious Pulmonary Complications of Human Immunodeficiency Virus Infection.
- Presentation of the Founders' Medal Award for 2014.
- Metabolomics of Bronchoalveolar Lavage Differentiate Healthy HIV-1-Infected Subjects from Controls.
- Aging promotes pro-fibrotic matrix production and increases fibrocyte recruitment during acute Lung injury.
- Activating the Nrf2-mediated antioxidant response element restores barrier function in the alveolar epithelium of HIV-1 transgenic rats.
- Chronic Alcohol Ingestion Primes the Lung for Bleomycin-Induced Fibrosis in Mice.
- Alcoholism Causes Alveolar Macrophage Zinc Deficiency and Immune Dysfunction.
- Chronic alcohol ingestion in rats decreases krüppel-like factor 4 expression and intracellular zinc in the Lung.
- Nicotinic Acetylcholine Receptors are Sensors for Ethanol in Lung Fibroblasts.
- Human immunodeficiency virus-1 transgene expression increases Pulmonary vascular resistance and exacerbates hypoxia-induced Pulmonary hypertension development.
- Alcohol causes alveolar epithelial oxidative stress by inhibiting the Nrf2/ARE signaling pathway.
- Alcohol Abuse, the Alveolar Macrophage and Pneumonia.
- HIV-1 transgene expression in rats induces differential expression of tumor necrosis factor alpha and zinc transporters in the liver and the Lung.
- Chronic Alcohol Ingestion Exacerbates Lung Epithelial Barrier Dysfunction in HIV-1 Transgenic Rats.
- Zinc Supplementation Restores PU.1 and Nrf2 Nuclear Binding in Alveolar Macrophages and Improves Redox Balance and Bacterial Clearance in the Lungs of Alcohol-Fed Rats.
- Alterations in the immuno-skeletal interface drive bone destruction in HIV-1 transgenic rats.
- Zinc deficiency mediates alcohol-induced alveolar epithelial and macrophage dysfunction in rats.
- HIV-1-transgene expression in rats decreases alveolar macrophage zinc levels and phagocytosis.
- Vascular oxidative stress and nitric oxide depletion in HIV-1 transgenic rats are reversed by glutathione restoration.
- Oxidant-induced atrogin-1 and transforming growth factor-beta1 precede alcohol-related myopathy in rats.
- Alcohol ingestion by donors amplifies experimental airway Disease after heterotopic transplantation.
- Alveolar type II cells from ethanol-fed rats produce a fibronectin-enriched extracellular matrix that promotes monocyte activation.
- Chronic alcohol ingestion alters claudin expression in the alveolar epithelium of rats.
- The alcoholic Lung: epidemiology, pathophysiology, and potential therapies.
- GM-CSF receptor expression and signaling is decreased in Lungs of ethanol-fed rats.
- Integrating acute Lung injury and regulation of alveolar fluid clearance.
- Cytokines and alcohol.
- Chronic ethanol ingestion increases superoxide production and NADPH oxidase expression in the Lung.
- Chronic ethanol ingestion in rats decreases granulocyte-macrophage colony-stimulating factor receptor expression and downstream signaling in the alveolar macrophage.
- Angiotensin II mediates glutathione depletion, transforming growth factor-beta1 expression, and epithelial barrier dysfunction in the alcoholic rat Lung.
- Mechanical ventilation exacerbates alveolar macrophage dysfunction in the Lungs of ethanol-fed rats.
- Transforming growth factor beta1 expression and activation is increased in the alcoholic rat Lung.
- Granulocyte/macrophage colony-stimulating factor treatment improves alveolar epithelial barrier function in alcoholic rat Lung.
- Chronic ethanol ingestion increases expression of the angiotensin II type 2 (AT2) receptor and enhances tumor necrosis factor-alpha- and angiotensin II-induced cytotox...
- Pulmonary alveolar proteinosis mimicking idiopathic Pulmonary fibrosis.
- Chronic alcohol abuse is associated with an increased incidence of acute respiratory distress syndrome and severity of multiple organ dysfunction in patients with sept...
- Mechanisms of alcohol-induced tissue injury.
- Chronic ethanol ingestion increases susceptibility to acute Lung injury: role of oxidative stress and tissue remodeling.
- Glutathione replacement preserves the functional surfactant phospholipid pool size and decreases sepsis-mediated Lung dysfunction in ethanol-fed rats.
- Anti-Retroviral Therapy Is Associated with Decreased Alveolar Glutathione Levels Even in Healthy HIV-Infected Individuals.
- Procysteine increases alcohol-depleted glutathione stores in rat plantaris following a period of abstinence.
- Alcohol abuse and acute Lung injury: can we target therapy?
- A sigh of relief about treating influenza in individuals with alcohol-use disorders?
- Alcohol primes the airway for increased interleukin-13 signaling.
- HIV-1 transgene expression in rats causes oxidant stress and alveolar epithelial barrier dysfunction.
- Procysteine stimulates expression of key anabolic factors and reduces plantaris atrophy in alcohol-fed rats.
- Na,K-ATPase expression is increased in the Lungs of alcohol-fed rats.
- Effect of HIV-1-related protein expression on cardiac and skeletal muscles from transgenic rats.
- Alcohol abuse and acute Lung injury: epidemiology and pathophysiology of a recently recognized association.
- Chronic ethanol ingestion increases aortic endothelial nitric oxide synthase expression and nitric oxide production in the rat.
- Molecular pathology and clinical aspects of alcohol-induced tissue injury.
- Influenza virus inhibits ENaC and Lung fluid clearance.
- Ethanol stimulates the expression of fibronectin in Lung fibroblasts via kinase-dependent signals that activate CREB.
- The effect of Chronic alcohol abuse on the incidence of ARDS and the severity of the multiple organ dysfunction syndrome in adults with septic shock: an interim and mu...
- Ethanol ingestion increases activation of matrix metalloproteinases in rat Lungs during acute endotoxemia.
- Mitochondrial antioxidant function is a potential mechanism for organ differences in interleukin-1-mediated tolerance to oxidative injury.
- The effects of Chronic alcohol abuse on pulmonary glutathione homeostasis.
- Ethanol ingestion via glutathione depletion impairs alveolar epithelial barrier function in rats.
- Diabetic patients have a decreased incidence of acute Respiratory distress syndrome.
- Mitochondrial glutathione replacement restores surfactant synthesis and secretion in alveolar epithelial cells of ethanol-fed rats.
- Chronic ethanol ingestion potentiates TNF-alpha-mediated oxidative stress and apoptosis in rat type II cells.
- Effect of Chronic ethanol ingestion on alveolar type II cell: glutathione and inflammatory mediator-induced apoptosis.
- Hypoxia injures endothelial cells by increasing endogenous xanthine oxidase activity.
- Tungsten treatment prevents tumor necrosis factor-induced injury of brain endothelial cells.
- Inactivation of xanthine oxidase by hydrogen peroxide involves site-directed hydroxyl radical formation.
- Inhaled nitric oxide prevents neutrophil-mediated, oxygen radical-dependent leak in isolated rat Lungs.
- Mitochondrial respiration scavenges extramitochondrial superoxide anion via a nonenzymatic mechanism.
- Phosphatidic acid signaling mediates Lung cytokine expression and Lung inflammatory injury after hemorrhage in mice.
- Interleukin-1 treatment increases neutrophils but not antioxidant enzyme activity or resistance to ischemia-reperfusion injury in rat kidneys.
- Intrinsic 5-lipoxygenase activity is required for neutrophil responsivity.
- Intratracheal but not intravascular interleukin-1 causes acute edematous injury in isolated neutrophil-perfused rat Lungs through an oxygen radical-mediated mechanism.
- Absence of electron transport (Rho 0 state) restores growth of a manganese-superoxide dismutase-deficient Saccharomyces cerevisiae in hyperoxia. Evidence for electron ...
- Endotoxin treatment increases Lung mitochondrial scavenging of extramitochondrial superoxide in hyperoxia-exposed rats.
- Inhaled NO prevents IL-1-induced neutrophil accumulation and associated acute edema in isolated rat Lungs.
- Modulating phosphatidic acid metabolism decreases oxidative injury in rat Lungs.
- In vitro selection of RNA-based irreversible inhibitors of human neutrophil elastase.
- Chronic ethanol ingestion impairs alveolar type II cell glutathione homeostasis and function and predisposes to endotoxin-mediated acute edematous Lung injury in rats.
- Endotoxin pretreatment in vivo increases the mitochondrial Respiratory capacity in rat hepatocytes.
- Receptor for advanced glycation end products in donor Lungs is associated with primary graft dysfunction after Lung transplantation.
- Ethanol ingestion impairs alveolar epithelial glutathione homeostasis and function, and predisposes to endotoxin-mediated acute Lung injury.
- Interleukin-1-induced tolerance to IL-1-induced oxidative Lung injury in rats.
- Chronic Alcohol Ingestion Increases Mortality and Organ Injury in a Murine Model of Septic Peritonitis.
- Alcohol ingestion disrupts alveolar epithelial barrier function by activation of macrophage-derived transforming growth factor beta1.
Schools
University of Mi Med Sch
Procedures Preformed
- Thoracentesis
- Ventilation Management
Conditions Treated
- Acidosis
- Alkalosis
- Asthma
- Bronchiectasis
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Doctors Specialties
- Critical Care Medicine
- Pulmonology
- Internal Medicine
Accepted Insurances
Awards
- Recipient of the Silver Pear Mentoring Award from the Department of Medicine
- Fellow
- Recipient of the Golden Apple Award for Teaching Excellence by the Internal Medicine housestaff
- Named one of the Best Doctors in America
- Pulmonary Star Award as the outstanding academic faculty member in the Division of Pulmonary, Allergy, & critical Care Medicine
- Department of Medicine Housestaff Teaching Award
- Young Faculty Research Award, Pulmonary Medicine, Southern Section
- Young Investigator Award, Western Section
- Edward Livingston Trudeau Scholar
- Trainee Travel Award
- Research Award
- Research Fellowship
Education
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University of Colorado -
University of Minnesota -
University of Michigan Medical School
Hospital
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Emory University Hospital -
Wesley Woods Center of Emory University -
Grady Memorial Hospital
Drug Facts
| NPI NUMBER | 1144258369 | |
| NPPES Provider LastName | GUIDOT | |
| NPPES Provider FirstName | DAVID | |
| NPPES Provider ZIPCode | 303221047 | |
| NPPES Provider State | GA | |
| Specialty Description | Pulmonary Disease | |
| Total Claim Count | 319.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1144258369 |
| Last Name Of The Provider | GUIDOT |
| First Name Of The Provider | DAVID |
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