USC Department of Dermatology
Dermatology
Dr. David T. Woodley, MD
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David Woodley is an expert in the field of Dermatology. He attended University of Nebraska Medical Center. University of Nebraska Medical Center is one of the top medical schools in the US. University of Nebraska Medical Center is ranked 60/4 in Research/PrimaryCare. He received 8 awards: "America's Top Doctors", "Top Doctors:LA Area", "Top Doctors:Southern California", "Elected Member", "Fellow (FASDS)", "Super Doctor", "Fellow (FAAD)" and "Top MD". David Woodley is a published physician as well. He has 53 publications published. The latest was: Breast Cancer MDA-MB-231 Cells Use Secreted Heat Shock Protein-90alpha (Hsp90) to Survive a Hostile Hypoxic Environment. He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Intravenously Administered Recombinant Human Type VII Collagen Derived from Chinese Hamster Ovary Cells Reverses the Disease Phenotype in Recessive Dystrophic Epidermo...
- A multicentre randomized trial of the treatment of patients with Pemphigus vulgaris with infliximab and prednisone compared with prednisone alone.
- Definitions and outcome measures for mucous membrane Pemphigoid: Recommendations of an international panel of experts.
- Treatment of fibroelastolytic papulosis with fractionated carbon dioxide laser.
- Aminoglycosides Restore Full-length Type VII Collagen by Overcoming Premature Termination Codons: Therapeutic Implications for Dystrophic Epidermolysis Bullosa.
- Omalizumab therapy for bullous Pemphigoid.
- De Novo Anti-Type VII Collagen Antibodies in Patients with Recessive Dystrophic Epidermolysis Bullosa.
- Topical Application of Recombinant Type VII Collagen Incorporates Into the Dermal-Epidermal Junction and Promotes Wound Closure.
- ExtraCellular Hsp90 (eHsp90) as the Actual Target in Clinical Trials: Intentionally or Unintentionally.
- Intravenously Injected Recombinant Human Type VII Collagen Homes to Skin Wounds and Restores Skin Integrity of Dystrophic Epidermolysis Bullosa.
- A potentially common peptide target in secreted heat shock protein-90alpha (Hsp90?) for hypoxia-inducible factor-1alpah (HIF-1?)-positive tumors.
- Epidermolysis bullosa acquisita.
- A fragment of secreted Hsp90? carries properties that enable it to accelerate effectively both acute and diabetic wound healing in mice.
- Epidermolysis bullosa acquisita: Autoimmunity to anchoring fibril collagen.
- Bone marrow transplantation for recessive dystrophic epidermolysis bullosa.
- Non-compensating roles between Nckalpha and Nckbeta in PDGF-BB signaling to promote human dermal fibroblast migration.
- Potential new insight into the pathogenesis of Psoriasis.
- Injection of recombinant human type VII collagen corrects the disease phenotype in a murine model of dystrophic epidermolysis bullosa.
- The role of oxygen in wound healing: a review of the literature.
- Profiling motility signal-specific genes in primary human keratinocytes.
- Characterization of molecular mechanisms underlying mutations in dystrophic epidermolysis bullosa using site-directed mutagenesis.
- Transforming growth factor alpha (TGFalpha)-stimulated secretion of HSP90alpha: using the receptor LRP-1/CD91 to promote human skin Cell migration against a TGFbeta-ri...
- Hypoxia induces epidermal keratinocyte matrix metalloproteinase-9 secretion via the protein kinase C pathway.
- Duplicating Autoimmune Bullous Diseases by Passively Transferring Autoantibodies into Animals.
- Familial epidermolysis bullosa acquisita.
- Autoimmunity to type VII collagen: epidermolysis bullosa acquisita.
- The cartilage matrix protein subdomain of type VII collagen is pathogenic for epidermolysis bullosa acquisita.
- Intravenously injected human fibroblasts home to Skin wounds, deliver type VII collagen, and promote wound healing.
- The role of IgE anti-basement membrane zone autoantibodies in bullous Pemphigoid.
- Transforming growth factor-alpha: a major human serum factor that promotes human keratinocyte migration.
- PKCdelta clustering at the leading edge and mediating growth factor-enhanced, but not ecm-initiated, dermal fibroblast migration.
- Induction of epidermolysis bullosa acquisita in mice by passive transfer of autoantibodies from patients.
- Fibroblasts as target Cells for DEB gene therapy.
- A "traffic control" role for TGFbeta3: orchestrating dermal and epidermal Cell motility during wound healing.
- Pterygium inversum unguis associated with stroke.
- Evidence that anti-type VII collagen antibodies are pathogenic and responsible for the clinical, histological, and immunological features of epidermolysis bullosa acqu...
- Wound-healing perspectives.
- Signals that initiate, augment, and provide directionality for human keratinocyte motility.
- Intradermal injection of lentiviral vectors corrects regenerated human dystrophic epidermolysis bullosa Skin tissue in vivo.
- Injection of recombinant human type VII collagen restores collagen function in dystrophic epidermolysis bullosa.
- Epidermolysis bullosa: then and now.
- A patient with both bullous Pemphigoid and epidermolysis bullosa acquisita: an example of intermolecular epitope spreading.
- Mechanism of human dermal fibroblast migration driven by type I collagen and platelet-derived growth factor-BB.
- Normal and gene-corrected dystrophic epidermolysis bullosa fibroblasts alone can produce type VII collagen at the basement membrane zone.
- Restoration of type VII collagen expression and function in dystrophic epidermolysis bullosa.
- The epidermolysis bullosa acquisita antigen (type VII collagen) is present in human colon and patients with crohn's disease have autoantibodies to type VII collagen.
- Drug-induced epidermolysis bullosa acquisita with antibodies to type VII collagen.
- The first international consensus on mucous membrane Pemphigoid: definition, diagnostic criteria, pathogenic factors, medical treatment, and prognostic indicators.
- An important role for protein kinase C-delta in human keratinocyte migration on dermal collagen.
- The recombinant expression of full-length type VII collagen and characterization of molecular mechanisms underlying dystrophic epidermolysis bullosa.
- Identification of the critical therapeutic entity in secreted hsp90α that promotes wound healing in newly re-standardized healthy and diabetic pig models.
- Breast Cancer MDA-MB-231 Cells Use Secreted Heat Shock Protein-90alpha (Hsp90α) to Survive a Hostile Hypoxic Environment.
Schools
University Of MissouriColumbia School Of Medicine
University Of Nc Hosps
University Of Ne College Of Med
Procedures Preformed
- Adjacent Tissue Transfer
- Destruction of Benign Skin Lesion
- Excision of Malignant Skin Lesion
- Mohs Surgery
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Conditions Treated
- Actinic Keratosis
- Athlete's Foot
- Basal Cell Carcinoma
- Benign Skin Lesion
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Doctors Specialties
- Dermatology
Accepted Insurances
Awards
- America's Top Doctors
- Top Doctors:LA Area
- Top Doctors:Southern California
- Elected Member
- Fellow (FASDS)
- Super Doctor
- Fellow (FAAD)
- Top MD
Education
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University of North Carolina Hospitals -
University of Nebraska Medical Center -
University of Missouri-Columbia School of Medicine
Hospital
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Children's Hospital Los Angeles -
White Memorial Medical Center -
Keck Hospital of USC
Drug Facts
| NPI NUMBER | 1871695551 | |
| NPPES Provider LastName | WOODLEY | |
| NPPES Provider FirstName | DAVID | |
| NPPES Provider ZIPCode | 900335310 | |
| NPPES Provider State | CA | |
| Specialty Description | Dermatology | |
| Total Claim Count | 451.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1871695551 |
| Last Name Of The Provider | WOODLEY |
| First Name Of The Provider | DAVID |
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