Assistant Professor, Pediatrics, Indiana University School of Medicine
Pediatric Hematology & Oncology
Dr. Grzegorz Nalepa, MD
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Grzegorz Nalepa is an expert in the field of Pediatric Hematology & Oncology. He went to Indiana University School of Medicine. Indiana University School of Medicine is ranked 45/47 in Research/PrimaryCare. He has 11 awards "SPR David G. Nathan Award in Basic Research", "Morris Green Academic Scholarship, Indiana University School of Medicine", "Red Shoes Award, Riley Hospital for Children", "AAP Integrated Research Pathway (Pediatric Residency and Fellowship)", "The Claude W. Smith Fellowship Award for Outstanding Research Performance", "The Professor John F. Trentin Scholarship Award for Academic Excellence", "Rockwell Scholarship Award", "The Deans Award for Best Medical Graduate", "Polish Ministry of Health Outstanding Medical Student Scholarship", "Third Best Scientific Presentation, Hanseatic Students Days of Science, Germany" and "Silver Medal". Grzegorz Nalepa has written publications. He has 21 publications published with PubMed. The lastest publication is: 'Leukemia and chromosomal instability in aged Fancc-/- mice.'
Publications
- Leukemia and chromosomal instability in aged Fancc-/- mice.
- Fanconi anemia signaling network regulates the spindle assembly checkpoint.
- The tumor suppressor CDKN3 controls mitosis.
- Normal hematopoiesis and neurofibromin-deficient Myeloproliferative disease require Erk.
- Imatinib mesylate for plexiform neurofibromas in patients with neurofibromatosis type 1: a phase 2 trial.
- The haploinsufficient hematopoietic microenvironment is critical to the pathological fracture repair in murine models of neurofibromatosis type 1.
- Genetic disruption of both Fancc and Fancg in mice recapitulates the hematopoietic manifestations of Fanconi anemia.
- The tumor suppressor CYLD regulates entry into mitosis.
- A functional genomic screen identifies a role for TAO1 kinase in spindle-checkpoint signalling.
- Anaphase initiation is regulated by antagonistic ubiquitination and deubiquitination activities.
- Drug discovery in the ubiquitin-proteasome system.
- Identification and functional evaluation of cellular and viral factors involved in the alteration of nuclear architecture during herpes simplex virus 1 infection.
- Recognition of phosphodegron motifs in human cyclin E by the SCF(Fbw7) ubiquitin ligase.
- Visualization of a highly organized intranuclear network of filaments in living mammalian cells.
- SCFbeta-TRCP links Chk1 signaling to degradation of the Cdc25A protein phosphatase.
- The cyclin E/Cdk2 substrate p220(NPAT) is required for S-phase entry, histone gene expression, and Cajal body maintenance in human somatic cells.
- Therapeutic anti-Cancer targets upstream of the proteasome.
- [Caspases and apoptosis: die and let live].
- Efp: a ring of independence?
- Fanconi anemia and the cell cycle: new perspectives on aneuploidy.
Conditions Treated
- Acute Leukemia
- Immune Thrombocytopenic Purpura (ITP)
- Liver Cancer
- Lung Cancer
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Doctors Specialties
- Pediatric Hematology
- Oncology
Accepted Insurances
Awards
- SPR David G. Nathan Award in Basic Research
- Morris Green Academic Scholarship, Indiana University School of Medicine
- Red Shoes Award, Riley Hospital for Children
- AAP Integrated Research Pathway (Pediatric Residency and Fellowship)
- The Claude W. Smith Fellowship Award for Outstanding Research Performance
- The Professor John F. Trentin Scholarship Award for Academic Excellence
- Rockwell Scholarship Award
- The Dean’s Award for Best Medical Graduate
- Polish Ministry of Health Outstanding Medical Student Scholarship
- Third Best Scientific Presentation, Hanseatic Students’ Days of Science, Germany
- Silver Medal
Education
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Indiana University School of Medicine -
Harvard Medical School -
Baylor College of Medicine -
Medical University of Silesia
Hospital
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Riley Hospital for Children at Indiana University Health
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