Professor, Neurology, Emory University School of Medicine
Neurology
Dr. Hyder A. Jinnah, MD
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Hyder Jinnah practices Neurology. He went to University of California San Diego School of Medicine. University of California San Diego School of Medicine is ranked 17 in research and 19 in primary care medicine Hyder Jinnah is a published doctor as well. He has 101 publications published. The latest was: Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtin. Doctor Hyder A. Jinnah, MD accepts Medicare.
Publications
- A clinical trial of safety and tolerability for the selective dopamine D1 receptor antagonist ecopipam in patients with Lesch-Nyhan Disease.
- A new knock-in mouse model of l-DOPA-responsive Dystonia.
- Secured web-based video repository for multicenter studies.
- Consequences of impaired purine recycling on the proteome in a cellular model of Lesch-Nyhan Disease.
- Dystonia and cerebellar degeneration in the leaner mouse mutant.
- Why are voluntary head movements in cervical Dystonia slow?
- Diagnosis and treatment of Dystonia.
- Purinosome formation as a function of the cell cycle.
- Clinical severity in Lesch-Nyhan Disease: The role of residual enzyme and compensatory pathways.
- Temporal profile of improvement of tardive dystonia after globus pallidus deep Brain stimulation.
- Treatment of myoclonus-Dystonia syndrome with tetrabenazine.
- Consequences of Delayed Dental Extraction in Lesch-Nyhan Disease.
- Globus pallidus deep Brain stimulation for adult-onset axial dystonia.
- Genotypic and phenotypic spectrum in attenuated variants of Lesch-Nyhan Disease.
- A rare sequence variant in intron 1 of THAP1 is associated with primary Dystonia.
- Genotype-phenotype correlations in neurogenetics: Lesch-Nyhan Disease as a model disorder.
- Transcriptomic approach to lesch-nyhan Disease.
- An (18)F-FDG PET study of cervical muscle in parkinsonian anterocollis.
- Dystonia as a network disorder: What is the role of the cerebellum?
- Designing Clinical Trials for Dystonia.
- Subtle microstructural changes of the cerebellum in a knock-in mouse model of DYT1 Dystonia.
- Phenotypic variation among seven members of one family with deficiency of hypoxanthine-guanine phosphoribosyltransferase.
- How long does it take to diagnose cervical Dystonia?
- Keeping your head on target.
- Purine metabolism during neuronal differentiation: the relevance of purine synthesis and recycling.
- The focal Dystonias: Current views and challenges for future research.
- The Dystonias: Past, present, and future.
- The effects of sapropterin on urinary monoamine metabolites in phenylketonuria.
- Clinical Reasoning: a 57-year-old man with jaw spasms.
- Neuropathology of cervical Dystonia.
- Personalized Chemotherapy Profiling Using Cancer Cell Lines from Selectable Mice.
- Stress, caffeine and ethanol trigger transient neurological dysfunction through shared mechanisms in a mouse calcium channelopathy.
- Subtle microstructural changes of the striatum in a DYT1 knock-in mouse model of Dystonia.
- Metabolic Disorders of purine metabolism affecting the nervous system.
- Delivery of neuroactive compounds to the Brain: potential utility of genetically modified cells.
- Grafting fibroblasts genetically modified to produce L-dopa in a rat model of Parkinson Disease.
- Animal models of Lesch-Nyhan Syndrome.
- Grafting genetically modified cells to the Brain: conceptual and technical issues.
- Amphetamine-induced behavioral phenotype in a hypoxanthine-guanine phosphoribosyltransferase-deficient mouse model of Lesch-Nyhan Syndrome.
- Survival and function of intrastriatally grafted primary fibroblasts genetically modified to produce L-dopa.
- Spontaneous locomotor hyperactivity in a mouse mutant with a deletion including the Snap gene on chromosome 2.
- Functional analysis of Brain dopamine systems in a genetic mouse model of Lesch-Nyhan syndrome.
- Animal models of generalized Dystonia.
- Behavioral aspects of Lesch-Nyhan Disease and its variants.
- Delineation of the motor disorder of Lesch-Nyhan Disease.
- Nifedipine suppresses self-injurious behaviors in animals.
- Sudden death in Lesch-Nyhan Disease.
- The role of dopamine receptors in the neurobehavioral Syndrome provoked by activation of L-type calcium channels in rodents.
- Lesch-Nyhan Disease: from mechanism to model and back again.
- Research priorities in spasmodic dysphonia.
- Cerebral ischemia mediates the effect of serum uric acid on cognitive function.
- The basal ganglia and cerebellum interact in the expression of dystonic movement.
- Experimental therapeutics for Dystonia.
- Consequences of impaired purine recycling in dopaminergic neurons.
- Paroxysmal dyskinesias in mice.
- Serum uric acid and Brain ischemia in normal elderly adults.
- Irregularity distinguishes limb tremor in cervical Dystonia from essential tremor.
- Neuroanatomical substrates for paroxysmal dyskinesia in lethargic mice.
- A human neuronal tissue culture model for Lesch-Nyhan Disease.
- Basal ganglia dopamine loss due to defect in purine recycling.
- Mechanisms for phenotypic variation in Lesch-Nyhan Disease and its variants.
- Neuronal voltage-gated calcium channels: brief overview of their function and clinical implications in neurology.
- Definition and classification of hyperkinetic movements in childhood.
- Environmental neurotoxin-induced progressive model of parkinsonism in rats.
- Attenuated variants of Lesch-Nyhan Disease.
- Kinematic and electromyographic tools for characterizing movement Disorders in mice.
- Treatment strategies for Dystonia.
- Localization of hypoxanthine-guanine phosphoribosyltransferase mRNA in the mouse Brain by in situ hybridization.
- Severe gouty arthritis and mild neurologic symptoms due to F199C, a newly identified variant of the hypoxanthine guanine phosphoribosyltransferase.
- Single-dose administration of MK-0657, an NR2B-selective NMDA antagonist, does not result in clinically meaningful improvement in motor function in patients with moder...
- Gene expression and mRNA editing of serotonin receptor 2C in Brains of HPRT gene knock-out mice, an animal model of Lesch-Nyhan disease.
- Hypoxanthine-guanine phosphoribosyl transferase regulates early developmental programming of dopamine neurons: implications for Lesch-Nyhan Disease pathogenesis.
- A Golgi study of neuronal architecture in a genetic mouse model for Lesch-Nyhan Disease.
- Lesch-Nyhan Disease in a female with a clinically normal monozygotic twin.
- Wilson''s Disease presenting with an unusual cough.
- Rodent models for Dystonia research: characteristics, evaluation, and utility.
- The motor disorder of classic Lesch-Nyhan Disease.
- The spectrum of mutations causing HPRT deficiency: an update.
- Abnormal motor behavior and vestibular dysfunction in the stargazer mouse mutant.
- Tetrahydrobiopterin deficiency and dopamine loss in a genetic mouse model of Lesch-Nyhan Disease.
- Expression of c-FOS in the Brain after activation of L-type calcium channels.
- Self-biting induced by activation of L-type calcium channels in mice: dopaminergic influences.
- Self-biting induced by activation of L-type calcium channels in mice: serotonergic influences.
- Paroxysmal dyskinesias in the lethargic mouse mutant.
- Abnormal cerebellar signaling induces Dystonia in mice.
- Triggers of paroxysmal dyskinesia in the calcium channel mouse mutant tottering.
- Pharmacologic thresholds for self-injurious behavior in a genetic mouse model of Lesch-Nyhan Disease.
- Oxidative stress and dopamine deficiency in a genetic mouse model of Lesch-Nyhan Disease.
- Neurocognitive functioning in Lesch-Nyhan Disease and partial hypoxanthine-guanine phosphoribosyltransferase deficiency.
- Phenylketonuria presenting in adulthood as progressive spastic paraparesis with dementia.
- Distinct behavioral and neuropathological abnormalities in transgenic mouse models of HD and DRPLA.
- Ocular motor dysfunction in Lesch-Nyhan Disease.
- Self-injurious behavior: gene-Brain-behavior relationships.
- The spectrum of inherited mutations causing HPRT deficiency: 75 new cases and a review of 196 previously reported cases.
- Calcium channel agonists and Dystonia in the mouse.
- Lesch-Nyhan Disease and the basal ganglia.
- Calcium channel activation and self-biting in mice.
- Bone marrow transplantation does not ameliorate the neurologic symptoms in mice deficient in hypoxanthine guanine phosphoribosyl transferase (HPRT).
- Intranuclear inclusions and neuritic aggregates in transgenic mice expressing a mutant N-terminal fragment of huntingtin.
- Clinimetric testing of the comprehensive cervical Dystonia rating scale.
Schools
University of California At San Diego
Procedures Preformed
- EMG (Electromyography)
- Non-Cosmetic Chemodenervation
Conditions Treated
- Brain Aneurysm
- Cerebral Artery Thrombosis
- Diplopia
- Dystonia
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Doctors Specialties
- Neurology
Accepted Insurances
Education
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Johns Hopkins University -
Penn State University -
University of California San Diego School of Medicine
Hospital
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Emory University Hospital
Drug Facts
| NPI NUMBER | 1477500635 | |
| NPPES Provider LastName | JINNAH | |
| NPPES Provider FirstName | HYDER | |
| NPPES Provider ZIPCode | 303220001 | |
| NPPES Provider State | GA | |
| Specialty Description | Neurology | |
| Total Claim Count | 72.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1477500635 |
| Last Name Of The Provider | JINNAH |
| First Name Of The Provider | HYDER |
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