Physician
Infectious Disease
Dr. Irini Sereti, MD
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Irini Sereti is an expert in the field of Infectious Disease. Irini Sereti is also a published doctor. She has 84 paper published. The most recent paper is: Complement activation by HIV-1-infected target cells enhances IL-2-stimulated but not unstimulated ADCC activity mediated by peripheral blood mononuclear cells.
Publications
- HIV infection and immune activation: the role of coinfections.
- Immune activation and HIV: an enduring relationship.
- Repeated Cycles of Recombinant Human Interleukin-7 in HIV-Infected Patients with low CD4 T cell Reconstitution on Antiretroviral Therapy: Results of two Phase II Multi...
- Serious Non-AIDS Events: Therapeutic Targets of Immune Activation and Chronic Inflammation in HIV Infection.
- T-Cell Depletion in the Colonic Mucosa of Patients With Idiopathic CD4+ Lymphopenia.
- Heme Oxygenase-1 Regulation of Matrix Metalloproteinase-1 Expression Underlies Distinct Disease Profiles in Tuberculosis.
- Response to Bandera et al.
- Plasma interleukin-18 levels are a biomarker of innate immune responses that predict and characterize tuberculosis-associated immune reconstitution inflammatory syndrome.
- Initiation of ART during Early Acute HIV Infection Preserves Mucosal Th17 Function and Reverses HIV-Related Immune Activation.
- Monocyte-Activation Phenotypes Are Associated With Biomarkers of Inflammation and Coagulation in Chronic HIV Infection.
- Mycobacterial Antigen Driven Activation of CD14++CD16- Monocytes Is a Predictor of Tuberculosis-Associated Immune Reconstitution Inflammatory Syndrome.
- Quantifying and predicting the effect of exogenous interleukin-7 on CD4+ T cells in HIV-1 infection.
- Role of IL-6 in Mycobacterium avium-Associated Immune Reconstitution Inflammatory Syndrome.
- Immunologic predictors of coronary artery calcium progression in a contemporary HIV cohort.
- Evidence for innate immune system activation in HIV-1 infected elite controllers.
- Cerebrospinal fluid HIV-1 compartmentalization in a patient with AIDS and acute varicella-zoster virus meningomyeloradiculitis.
- Immune restoration after antiretroviral therapy: the pitfalls of hasty or incomplete repairs.
- Peptide library-based evaluation of T-cell receptor breadth detects defects in global and regulatory activation in human immunologic diseases.
- Interleukin-7 promotes HIV persistence during antiretroviral therapy.
- IL-7 induces expression and activation of integrin α4β7 promoting naive T-cell homing to the intestinal mucosa.
- Prevalence of Strongyloides stercoralis in an urban US AIDS cohort.
- Towards a scalable HIV cure research agenda: the role of co-infections.
- Cytomegalovirus immune reconstitution inflammatory syndrome manifesting as acute appendicitis in an HIV-infected patient.
- Paradoxical Tuberculosis Immune Reconstitution Inflammatory Syndrome (TB-IRIS) in HIV Patients with Culture Confirmed Pulmonary Tuberculosis in India and the Potential...
- Identification of dynamically distinct subpopulations of T lymphocytes that are differentially affected by HIV.
- CD4 T cell expansions are associated with increased apoptosis rates of T lymphocytes during IL-2 cycles in HIV infected patients.
- Interleukin-2 induced immune effects in human immunodeficiency virus-infected patients receiving intermittent interleukin-2 immunotherapy.
- Disseminated nocardiosis in a patient with X-linked chronic granulomatous disease and human immunodeficiency virus infection.
- Immunopathogenesis of human immunodeficiency virus: implications for immune-based therapies.
- Alopecia universalis and Graves'' disease in the setting of immune restoration after highly active antiretroviral therapy.
- Interleukin 2 leads to dose-dependent expression of the alpha chain of the IL-2 receptor on CD25-negative T lymphocytes in the absence of exogenous antigenic stimulation.
- Complement activation by HIV-1-infected target cells enhances IL-2-stimulated but not unstimulated ADCC activity mediated by peripheral blood mononuclear cells.
- Cycling of gut mucosal CD4+ T cells decreases after prolonged anti-retroviral therapy and is associated with plasma LPS levels.
- Immune reconstitution inflammatory syndrome: the trouble with immunity when you had none.
- Decreases in IL-7 levels during antiretroviral treatment of HIV infection suggest a primary mechanism of receptor-mediated clearance.
- Lack of compartmentalization of HIV-1 quasispecies between the gut and peripheral blood compartments.
- Higher levels of CRP, D-dimer, IL-6, and hyaluronic acid before initiation of antiretroviral therapy (ART) are associated with increased risk of AIDS or death.
- Differential effects of HIV viral load and CD4 count on proliferation of naive and memory CD4 and CD8 T lymphocytes.
- CD4+ T Cells, Including Th17 and Cycling Subsets, are Intact in the Gut Mucosa of HIV-1 Infected Long-Term Non- Progressors.
- Transmitted raltegravir resistance in an HIV-1 CRF_AG-infected patient.
- Markers of endothelial dysfunction, coagulation and tissue fibrosis independently predict venous thromboembolism in HIV.
- The power of 1 in HIV therapeutics.
- The effect of intermittent IL-2 therapy on CD4 T cells in the gut in HIV-1-infected patients.
- Plasma levels of soluble CD14 independently predict mortality in HIV infection.
- HIV-1 viruses detected during episodic blips following interleukin-7 administration are similar to the viruses present before and after interleukin-7 therapy.
- Biomarkers in immune reconstitution inflammatory syndrome: signals from pathogenesis.
- Detection of EBV genomes in plasmablasts/plasma cells and non-B cells in the blood of most patients with EBV lymphoproliferative disorders by using Immuno-FISH.
- The effect of leflunomide on cycling and activation of T-cells in HIV-1-infected participants.
- Elevated frequencies of highly activated CD4+ T cells in HIV+ patients developing immune reconstitution inflammatory syndrome.
- Th1-driven immune reconstitution disease in Mycobacterium avium-infected mice.
- Risk factor analyses for immune reconstitution inflammatory syndrome in a randomized study of early vs. deferred ART during an opportunistic infection.
- Traditional risk factors and D-dimer predict incident cardiovascular disease events in chronic HIV infection.
- Changing lanes in ICL.
- d-Dimer and CRP levels are elevated prior to antiretroviral treatment in patients who develop IRIS.
- Where have all the T cells gone?
- Interleukin-2 cycling causes transient increases in high-sensitivity C-reactive protein and D-dimer that are not associated with plasma HIV-RNA levels.
- Immunopathogenesis of asymptomatic chronic HIV Infection: the calm before the storm.
- Evidence for translocation of microbial products in patients with idiopathic CD4 lymphocytopenia.
- IL-7 administration drives T cell-cycle entry and expansion in HIV-1 infection.
- Inferiority of IL-2 alone versus IL-2 with HAART in maintaining CD4 T cell counts during HAART interruption: a randomized controlled trial.
- The common gamma-chain cytokines IL-2, IL-7, IL-15, and IL-21 induce the expression of programmed death-1 and its ligands.
- CD4 T cell survival after intermittent interleukin-2 therapy is predictive of an increase in the CD4 T cell count of HIV-infected patients.
- HIV infection and the gut: scarred for life?
- The effect of continuous versus pericycle antiretroviral therapy on IL-2 responsiveness.
- IL-15 acts as a potent inducer of CD4(+)CD25(hi) cells expressing FOXP3.
- Idiopathic CD4+ lymphocytopenia: natural history and prognostic factors.
- Effects of delays in peripheral blood processing, including cryopreservation, on detection of CD31 expression on naive CD4 T cells.
- CD4+ T cell responses to interleukin-2 administration in HIV-infected patients are directly related to the baseline level of immune activation.
- Decreased CD127 expression on T Cells in HIV-1-infected adults receiving antiretroviral therapy with or without intermittent IL-2 therapy.
- Delayed sample processing leads to marked decreases in measured plasma IL-7 levels.
- Naïve T-cell dynamics in human immunodeficiency virus type 1 infection: effects of highly active antiretroviral therapy provide insights into the mechanisms of naive T...
- Interruption of antiretroviral therapy blunts but does not abrogate CD4 T-cell responses to interleukin-2 administration in HIV infected patients.
- Induction of prolonged survival of CD4+ T lymphocytes by intermittent IL-2 therapy in HIV-infected patients.
- In vivo expansion of CD4CD45RO-CD25 T cells expressing foxP3 in IL-2-treated HIV-infected patients.
- Graves' disease after interleukin-2 therapy in a patient with human immunodeficiency virus infection.
- Effects of lymphocyte isolation and timing of processing on detection of CD127 expression on T cells in human immunodeficiency virus-infected patients.
- BAY 50-4798, a novel, high-affinity receptor-specific recombinant interleukin-2 analog, induces dose-dependent increases in CD25 expression and proliferation among uns...
- IL-2-induced CD4+ T-cell expansion in HIV-infected patients is associated with long-term decreases in T-cell proliferation.
- A randomized, double-blinded, placebo-controlled trial of intermittent administration of interleukin-2 and prednisone in subjects infected with human immunodeficiency ...
- Incomplete CD4 T cell recovery in HIV-1 infection after 12 months of highly active antiretroviral therapy is associated with ongoing increased CD4 T cell activation an...
- Cutting edge: L-selectin (CD62L) expression distinguishes small resting memory CD4+ T cells that preferentially respond to recall antigen.
- Long-term effects of intermittent interleukin 2 therapy in patients with HIV infection: characterization of a novel subset of CD4(+)/CD25(+) T cells.
- Increased peripheral expansion of naive CD4+ T cells in vivo after IL-2 treatment of patients with HIV infection.
Doctors Specialties
- Infectious Disease Medicine
- Infectious Disease
Accepted Insurances
Education
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National Institutes of Health Clinical Center -
University of Athens
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