Professor, Dermatology, Indiana University School of Medicine
Dermatology
Dr. Jeffrey B. Travers, MD
|
|
Give Review
Jeffrey B. Travers, MD works in the field of Dermatology. He researched medicine at The Ohio State University College of Medicine. He over the years received 2 awards: "Elected Member" and "Fellow (FAAD)". Jeffrey Travers is a published physician as well. He has 102 publications published. The latest was: Identification and pharmacological characterization of platelet-activating factor and related 1-palmitoyl species in human inflammatory blistering diseases.
Publications
- Important implications and new uses of ablative lasers in dermatology: fractional carbon dioxide laser prevention of Skin cancer.
- Topical photodynamic therapy induces systemic immunosuppression via generation of platelet-activating factor receptor ligands.
- Trichodysplasia Spinulosa in Gorlin Syndrome Treated With Vismodegib-Reply.
- Chemotherapeutic agents subvert tumor immunity by generating agonists of platelet-activating factor.
- Development of trichodysplasia spinulosa: case report of a patient with gorlin syndrome treated with vismodegib.
- Toxic Interaction between Th2 Cytokines and Staphylococcus aureus in Atopic Dermatitis.
- Staphylococcus δ-toxin induces allergic skin disease by activating mast Cells.
- Topical Application of a Vitamin D Analogue Exacerbates Atopic Dermatitis and Induces the Atopic Dermatitis-like Phenotype in Stat6VT Mice.
- Exogenous melatonin reproduces the effects of short day lengths on hippocampal function in male white-Footed mice, Peromyscus leucopus.
- Familial granular Cell tumors: a case report and review of the literature.
- Cigarette Smoke Exposure Inhibits Contact Hypersensitivity via the Generation of Platelet-Activating Factor Agonists.
- Fibroblast Senescence and Squamous Cell Carcinoma: How Wounding Therapies Could Be Protective.
- Chloroquine Promotes Apoptosis in Melanoma Cells by Inhibiting BH3 domain Mediated PUMA Degradation.
- Imatinib mesylate for plexiform neurofibromas in patients with neurofibromatosis type 1: a phase 2 trial.
- Fractionated laser resurfacing corrects the inappropriate UVB response in geriatric Skin.
- The environmental stressor ultraviolet B radiation inhibits murine anti-tumor immunity through its ability to generate Platelet-activating factor agonists.
- Loss of the Platelet Activating Factor Receptor in mice augments PMA-induced inflammation and cutaneous chemical carcinogenesis.
- Platelet-activating Factor does not Mediate UVB-induced Local Immune Suppression.
- Treatment Outcomes of Secondarily Impetiginized Pediatric Atopic Dermatitis Lesions and the Role of Oral Antibiotics.
- Acquired STAT4 deficiency as a consequence of Cancer chemotherapy.
- Increased skin barrier disruption by sodium lauryl sulfate in mice expressing a constitutively active STAT6 in T Cells.
- Reversing the aging stromal phenotype prevents Carcinoma initiation.
- Progression of Toxic Epidermal Necrolysis After Tanning Bed Exposure.
- Dendritic cells produce inflammatory cytokines in response to bacterial products from Staphylococcus aureus-infected Atopic dermatitis lesions.
- Altered cytokine production by dendritic cells from infants with Atopic dermatitis.
- Identification of staphylococcal protein A in infected Atopic dermatitis lesions.
- Ultraviolet B radiation of human Skin generates platelet-activating factor receptor agonists.
- IL-4 regulates Skin homeostasis and the predisposition toward allergic Skin inflammation.
- Epidermal platelet-activating factor receptor activation and ultraviolet B radiation result in synergistic tumor necrosis factor-alpha production.
- Infected Atopic dermatitis lesions contain pharmacologic amounts of lipoteichoic acid.
- The peroxisome proliferator-activated receptor gamma system regulates ultraviolet B-induced prostaglandin e(2) production in human epidermal keratinocytes.
- cis-Urocanic acid stimulates primary human keratinocytes independently of serotonin or platelet-activating factor receptors.
- p21-activated kinase regulates mast Cell degranulation via effects on calcium mobilization and cytoskeletal dynamics.
- A new paradigm for the role of aging in the development of Skin cancer.
- Ultraviolet B radiation generated platelet-activating factor receptor agonist formation involves EGF-R-mediated reactive oxygen species.
- Involvement of platelet-activating factor in ultraviolet B-induced hyperalgesia.
- Oxidized glycerophosphocholines as biologically active mediators for ultraviolet radiation-mediated effects.
- UVB radiation-mediated inhibition of Contact hypersensitivity reactions is dependent on the platelet-activating factor system.
- UVB-induced senescence in human keratinocytes requires a functional insulin-like growth factor-1 receptor and p53.
- Augmentation of UVB radiation-mediated early gene expression by the epidermal platelet-activating factor receptor.
- Scratching the surface: towards understanding the pathogenesis of Atopic dermatitis.
- Vaccinia virus infection induces dendritic Cell maturation but inhibits antigen presentation by MHC class II.
- Ultraviolet B radiation generates platelet-activating factor-like phospholipids underlying cutaneous damage.
- Gene correction reduces cutaneous inflammation and granuloma formation in murine X-linked chronic granulomatous disease.
- Staphylococcal lipoteichoic acid inhibits delayed-type hypersensitivity reactions via the platelet-activating factor receptor.
- Epidermal peroxisome proliferator-activated receptor gamma as a target for ultraviolet B radiation.
- Moxifloxacin-associated drug hypersensitivity syndrome with toxic epidermal necrolysis and fulminant hepatic failure.
- Augmentation of chemotherapy-induced cytokine production by expression of the platelet-activating factor receptor in a human epithelial carcinoma Cell line.
- Amphiregulin overexpression results in rapidly growing keratinocytic tumors: an in vivo xenograft model of keratoacanthoma.
- Distinct patterns of gene expression in the skin lesions of Atopic dermatitis and psoriasis: a gene microarray analysis.
- Cytokine milieu of Atopic dermatitis, as compared to psoriasis, skin prevents induction of innate immune response genes.
- The epidermal platelet-activating factor receptor augments chemotherapy-induced apoptosis in human carcinoma Cell lines.
- Augmentation of staphylococcal alpha-toxin signaling by the epidermal platelet-activating factor receptor.
- Hyperoxia alters phorbol ester-induced phospholipase D activation in bovine lung microvascular endothelial Cells.
- Endothelial induction of the T-Cell chemokine CCL21 in T-Cell autoimmune diseases.
- Activation of epidermal vanilloid receptor-1 induces release of proinflammatory mediators in human keratinocytes.
- Identification of platelet-activating factor acetylhydrolase II in human Skin.
- Low-molecular-weight heparins inhibit CCL21-induced T Cell adhesion and migration.
- The platelet-activating factor receptor activates the extraCellular signal-regulated kinase mitogen-activated protein kinase and induces proliferation of epidermal cel...
- Enhanced cutaneous inflammatory reactions to Aspergillus fumigatus in a murine model of chronic granulomatous disease.
- Decreased IL-15 may contribute to elevated IgE and acute inflammation in Atopic dermatitis.
- Topical application of a platelet activating factor receptor agonist suppresses phorbol ester-induced acute and chronic inflammation and has Cancer chemopreventive act...
- Skin resurfacing procedures: new and emerging options.
- Systemic Platelet-activating Factor Receptor Activation Augments Experimental Lung Tumor Growth and Metastasis.
- STAT6-Mediated Keratitis and Blepharitis: A Novel Murine Model of Ocular Atopic Dermatitis.
- Beta-oxidation of 12(S)-hydroxy-5,8,10,14-eicosatetraenoic acid by MOLT-4 lymphocytes.
- Metabolism of 13-hydroxy-9,11-octadecadienoic acid by MOLT-4 lymphocytes.
- Taste reactivity and Fos expression in GAD1-EGFP transgenic mice.
- Mechanisms of exertional dyspnea in patients with Cancer.
- Platelet-activating factor stimulates phosphatidylinositol hydrolysis in the rat myometrium.
- Novel immunomodulators for topical Skin disease therapy.
- Metabolism of 15-hydroxy-5,8,11,13-eicosatetraenoic acid by MOLT-4 Cells and blood T-lymphocytes.
- Melanocyte mitogens induce both melanocyte chemokinesis and chemotaxis.
- The metabolism of platelet-activating factor in human T-lymphocytes.
- Differentiating keratinocytes express a novel cytochrome P450 enzyme, CYP2B19, having arachidonate monooxygenase activity.
- Binding of carbamyl-platelet-activating factor to the Raji lymphoblast platelet-activating factor receptor.
- Evidence for platelet-activating factor receptors in several B lymphoblastoid Cell lines.
- Lymphocyte activation in the pathogenesis of Psoriasis.
- Nf1+/- mast Cells induce neurofibroma like phenotypes through secreted TGF-beta signaling.
- Identification of functional platelet-activating factor receptors in Raji lymphoblasts.
- Activation of the epidermal platelet-activating factor receptor results in ICAM-1 expression.
- Genetic and biochemical evidence that haploinsufficiency of the Nf1 tumor suppressor gene modulates melanocyte and mast Cell fates in vivo.
- Hyperactivation of p21(ras) and the hematopoietic-specific Rho GTPase, Rac2, cooperate to alter the proliferation of neurofibromin-deficient mast Cells in vivo and in ...
- Evidence for a streptococcal superantigen-driven process in acute guttate Psoriasis.
- The role of superantigens in Skin disease.
- Metabolism of leukotriene B4 (LTB4) and 12-hydroxy-5,8,10,14-eicosatetraenoic acid (12-HETE) in human keratinocytes.
- Identification of sn-2 acetyl glycerophosphocholines in human keratinocytes.
- The keratinocyte as a target for staphylococcal bacterial toxins.
- Evidence for the involvement of the NADPH oxidase enzyme complex in the optimal accumulation of Platelet-activating factor in the human Cell line PLB-985.
- Oxidative stress can activate the epidermal platelet-activating factor receptor.
- Epidermal HLA-DR and the enhancement of cutaneous reactivity to superantigenic toxins in Psoriasis.
- The IGF-1/IGF-1R signaling axis in the Skin: a new role for the dermis in aging-associated Skin cancer.
- The role of superantigens in human diseases: therapeutic implications for the treatment of Skin diseases.
- Encoding a superantigen by Staphylococcus aureus does not affect clinical characteristics of infected Atopic dermatitis lesions.
- Platelet-activating factor biosynthesis induced by various stimuli in human HaCaT keratinocytes.
- The platelet-activating factor receptor protects epidermal Cells from tumor necrosis factor (TNF) alpha and TNF-related apoptosis-inducing ligand-induced apoptosis thr...
- Identification and pharmacological characterization of platelet-activating factor and related 1-palmitoyl species in human inflammatory blistering diseases.
- Evidence for involvement of the epidermal platelet-activating factor receptor in ultraviolet-B-radiation-induced interleukin-8 production.
- Use of enhanced green fluorescent protein to monitor retroviral-mediated gene therapy in human keratinocytes.
- Acute keratinocyte damage stimulates platelet-activating factor production.
- Examining the Differences in Current Regulatory Processes for Sunscreens and Proposed Safety Assessment Paradigm.
Schools
Ohio State University College Of Medicine and Public Health
Riverside Methodist Hospital
University Colo Health Science Center
National Jewish Center Immun and Resp Med
Procedures Preformed
- Destruction of Benign Skin Lesion
- Destruction of Malignant Skin Lesion
- Excision of Malignant Skin Lesion
Conditions Treated
- Actinic Keratosis
- Athlete's Foot
- Atopic Dermatitis (Eczema)
- Basal Cell Carcinoma
- View All
Doctors Specialties
- Dermatology
Accepted Insurances
Awards
- Elected Member
- Fellow (FAAD)
Education
-
University of Colorado -
Riverside Methodist Hospitals - OhioHealth -
The Ohio State University College of Medicine
Drug Facts
| NPI NUMBER | 1770599979 | |
| NPPES Provider LastName | TRAVERS | |
| NPPES Provider FirstName | JEFFREY | |
| NPPES Provider ZIPCode | 462025149 | |
| NPPES Provider State | IN | |
| Specialty Description | Dermatology | |
| Total Claim Count | 128.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1770599979 |
| Last Name Of The Provider | TRAVERS |
| First Name Of The Provider | JEFFREY |
| View All |
Similar Doctors
|
|
Dr. Stephen E. Wolverton, MD
 This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Kathryn A. Kroeger, MD
This Doctor is OpenDoctor Recommended.
|
|
Dr. Sally A. Booth, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Annette M. Dinneen, MD
This Doctor is OpenDoctor Recommended.
|
|
|
Dr. Katherine B. Lee, MD |
|
|
Dr. Thomas A. Graham, MD |
|
Veneda K. Vann, NP
This Doctor is OpenDoctor Recommended.
|