Associate Professor, Psychiatry, University of Iowa College of Medicine
Psychiatry
Dr. John A. Wemmie, MD
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Dr. John A. Wemmie is an expert in the field of Psychiatry. He attended University of Iowa Carver College of Medicine. University of Iowa Carver College of Medicine is ranked 29/16 in Research/PrimaryCare. He received 1 awards: "Elected Member". John Wemmie has written publications. He has 44 publications published with PubMed. The lastest paper is: 'Peripheral inflammation during abnormal mood states in bipolar I disorder.'
Publications
- Peripheral inflammation during abnormal mood states in bipolar I disorder.
- T1ÃÂ imaging in premanifest Huntington disease reveals changes associated with disease progression.
- Brain abnormalities in bipolar disorder detected by quantitative T1ÃÂ mapping.
- Quantitative T1ÃÂ mapping links the cerebellum and lithium use in bipolar disorder.
- Rapid acquisition strategy for functional T1ÃÂ mapping of the brain.
- Drug abuse and the simplest neurotransmitter.
- Protons are a neurotransmitter that regulates synaptic plasticity in the lateral amygdala.
- Acid-sensing ion channels contribute to synaptic transmission and inhibit cocaine-evoked plasticity.
- Acid-sensing ion channels 1a (ASIC1a) inhibit neuromuscular transmission in female mice.
- Regulator of G-protein signaling 6 (RGS6) promotes anxiety and depression by attenuating serotonin-mediated activation of the 5-HT1A receptor-adenylyl cyclase axis.
- Evaluation of activity-dependent functional pH and T1ÃÂ response in the visual cortex.
- Localization and Behaviors in Null Mice Suggest that ASIC1 and ASIC2 Modulate Responses to Aversive Stimuli.
- Functional T1ÃÂ Imaging in Panic Disorder.
- The non-canonical Wnt ligand Wnt5a rescues morphological deficits in Prickle2-deficient hippocampal neurons.
- Acid-sensing ion channels in pain and disease.
- Disruption of the non-canonical Wnt gene PRICKLE2 leads to autism-like behaviors with evidence for hippocampal synaptic dysfunction.
- Fear and panic in humans with bilateral amygdala damage.
- Maternal antioxidant blocks programmed cardiovascular and behavioral stress responses in adult mice.
- Neonatal leptin administration alters regional brain volumes and blocks neonatal growth restriction-induced behavioral and cardiovascular dysfunction in male mice.
- ASIC1 and ASIC3 play different roles in the development of Hyperalgesia after inflammatory muscle injury.
- Histologic examination of the eye of acid-sensing ion channel 1a knockout mice.
- The amygdala is a chemosensor that detects carbon dioxide and acidosis to elicit fear behavior.
- Role of calcitonin gene-related peptide in light-aversive behavior: implications for migraine.
- Acid-sensing ion channel-1a in the amygdala, a novel therapeutic target in depression-related behavior.
- Oxidant regulated inter-subunit disulfide bond formation between ASIC1a subunits.
- The cytoskeletal protein alpha-actinin regulates acid-sensing ion channel 1a through a C-terminal interaction.
- Restoring Acid-sensing ion channel-1a in the amygdala of knock-out mice rescues fear memory but not unconditioned fear responses.
- Seizure termination by acidosis depends on ASIC1a.
- Acid-sensing ion channel-1 contributes to axonal degeneration in autoimmune inflammation of the central nervous system.
- Targeting ASIC1a reduces innate fear and alters neuronal activity in the fear circuit.
- Acid-sensing ion channel 1a is a postsynaptic proton receptor that affects the density of dendritic spines.
- Functional correction of CNS phenotypes in a lysosomal storage disease model using adeno-associated virus type 4 vectors.
- Stomatin modulates gating of acid-sensing ion channels.
- The ion channel ASIC1 contributes to visceral but not cutaneous mechanoreceptor function.
- Subunit-dependent high-affinity zinc inhibition of acid-sensing ion channels.
- Neuroprotection in ischemia: blocking calcium-permeable acid-sensing ion channels.
- Acid-sensing ion channel 2 (ASIC2) modulates ASIC1 H+-activated currents in hippocampal neurons.
- Overexpression of acid-sensing ion channel 1a in transgenic mice increases acquired fear-related behavior.
- Acid-sensing ion channel 1 is localized in brain regions with high synaptic density and contributes to fear conditioning.
- ASIC3 and ASIC1 mediate FMRFamide-related peptide enhancement of H+-gated currents in cultured dorsal root ganglion neurons.
- cAMP-dependent protein kinase phosphorylation of the acid-sensing ion channel-1 regulates its binding to the protein interacting with C-kinase-1.
- The acid-activated ion channel ASIC contributes to synaptic plasticity, learning, and memory.
- Heteromultimers of DEG/ENaC subunits form H+-gated channels in mouse sensory neurons.
Schools
University of Ia Roy J amp L Carver Com
University Iowa Hosps and Clinics
Doctors Specialties
- Psychiatry
Accepted Insurances
Awards
- Elected Member
Education
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University of Iowa Hospitals and Clinics -
University of Iowa Carver College of Medicine
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