Professor of Medicine, Case West Res University
Nephrology
Dr. John R. Sedor, MD
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John Sedor works in the field of Nephrology. He graduated from University of Virginia School of Medicine. He over the years received 1 awards: "Top MD". John R. Sedor is a published doctor. He published 46 publications, including: 'Severe vascular calcification and tumoral calcinosis in a family with hyperphosphatemia: a fibroblast growth factor 23 mutation identified by exome sequencing.' He accepts Medicare.
Publications
- Severe vascular calcification and tumoral calcinosis in a family With hyperphosphatemia: a fibroblast growth factor 23 mutation identified by exome sequencing.
- The Kidney Research National Dialogue: Gearing Up to Move Forward.
- Apolipoprotein L1 gene variants associate with prevalent Kidney but not prevalent cardiovascular disease in the Systolic Blood Pressure Intervention Trial.
- Design of the Nephrotic Syndrome Study Network (NEPTUNE) to evaluate primary glomerular Nephropathy by a multidisciplinary approach.
- Family history and Kidney disease.
- WT1-interacting protein (Wtip) regulates podocyte phenotype by cell-cell and cell-matrix contact reorganization.
- APOL1 Localization in Normal Kidney and Nondiabetic Kidney Disease.
- A systems view of genetics in chronic Kidney disease.
- Use of diffusion tensor MRI to identify early changes in Diabetic nephropathy.
- Identification of the Nephropathy-Susceptibility Locus HIVAN4.
- The genetics of common Kidney disease: a pathway toward clinical relevance.
- Are cubilin (CUBN) variants at the Heart of urinary albumin excretion?
- Mesangial cell integrin αvβ8 provides glomerular endothelial cell cytoprotection by sequestering TGF-β and regulating PECAM-1.
- Podocyte injury induces nuclear translocation of WTIP via microtubule-dependent transport.
- Genetics and the Kidney: promise, potential, and challenges. Introduction.
- Translating associations between common Kidney diseases and genetic variation into the clinic.
- Uromodulin and translational medicine: will the SNPs bring zip to clinical practice?
- Out on a Lim: Chronic Kidney Disease, Podocyte Phenotype and the Wilm's Tumor Interacting Protein (WTIP).
- Tissue proteomics: a new investigative tool for Renal biopsy analysis.
- World Kidney Day 2009: problems and challenges in the emerging epidemic of Kidney disease.
- Hypertension-associated Kidney disease: perhaps no more.
- Chronic Kidney disease and its complications.
- Activation of adenosine 2A receptors preserves structure and function of podocytes.
- Risk factors for development and progression of diabetic Kidney disease and treatment patterns among diabetic siblings of patients with diabetic Kidney disease.
- Familial clustering of chronic Kidney disease.
- Mining the genome for susceptibility to Diabetic nephropathy: the role of large-scale studies and consortia.
- Math6 expression during Kidney development and altered expression in a mouse model of glomerulosclerosis.
- Beta8 integrin binds Rho GDP dissociation inhibitor-1 and activates Rac1 to inhibit mesangial cell myofibroblast differentiation.
- Frontiers in Diabetic nephropathy: can we predict who will get sick?
- Mechanical force-activated phospholipase D is mediated by Galpha12/13-Rho and calmodulin-dependent kinase in Renal epithelial cells.
- WT1-interacting protein and ZO-1 translocate into podocyte nuclei after puromycin aminonucleoside treatment.
- Association of metabolic syndrome in nondiabetic patients with increased risk for chronic Kidney disease: the fat lady sings.
- A dipstick protein and specific gravity algorithm accurately predicts pathological Proteinuria.
- Variants in the Wilms' tumor gene are associated With focal segmental glomerulosclerosis in the African American population.
- An activating mutant of Cdc42 that fails to interact With Rho GDP-dissociation inhibitor localizes to the plasma membrane and mediates actin reorganization.
- The metabolic syndrome as a risk factor for chronic Kidney disease: more than a fat chance?
- Renal phenotype is exacerbated in Os and lpr double mutant mice.
- The NHE1 Na+/H+ exchanger recruits ezrin/radixin/moesin proteins to regulate Akt-dependent cell survival.
- A WT1 co-regulator controls podocyte phenotype by shuttling between adhesion structures and nucleus.
- Linkage analysis of candidate loci for end-stage Renal disease due to diabetic nephropathy.
- Renal tubular epithelial cell apoptosis is associated With caspase cleavage of the NHE1 Na+/H+ exchanger.
- Serial analysis of gene expression.
- Fas activation induces Renal tubular epithelial cell beta 8 integrin expression and function in the absence of apoptosis.
- Approaches to understanding susceptibility to Nephropathy: from genetics to genomics.
- A Genome-Wide Search for Linkage of Estimated Glomerular Filtration Rate (eGFR) in the Family Investigation of Nephropathy and Diabetes (FIND).
Schools
Virginia Commonwealth University School Of Medicine
University Hosps Case West Res
Procedures Preformed
- Hemodialysis
Conditions Treated
- Acidosis
- Acute Kidney Failure
- Alkalosis
- Amyloidosis
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Doctors Specialties
- Nephrology
Accepted Insurances
Awards
- Top MD
Education
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Case Western Reserve University - University Hospitals -
University of Virginia School of Medicine
Hospital
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MetroHealth Medical Center
Drug Facts
| NPI NUMBER | 1497789887 | |
| NPPES Provider LastName | SEDOR | |
| NPPES Provider FirstName | JOHN | |
| NPPES Provider ZIPCode | 441091900 | |
| NPPES Provider State | OH | |
| Specialty Description | Nephrology | |
| Total Claim Count | 45.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1497789887 |
| Last Name Of The Provider | SEDOR |
| First Name Of The Provider | JOHN |
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