Physician
Rheumatology
Dr. Jon D. Levine, MD
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Jon D. Levine, MD is an expert in the field of Rheumatology. He researched medicine at University of California San Francisco School of Medicine. University of California San Francisco School of Medicine is a top medical university. University of California San Francisco School of Medicine has a rank of 3 in research and a 3 in primary care He has 2 awards "Elected Member" and "Fellow (FACR)". Dr. Jon D. Levine is a published doctor. He published 94 publications, including: 'Extracellular Matrix Hyaluronan Signals Via its CD44 Receptor in the Increased Responsiveness to Mechanical Stimulation.' Levine is registered with Medicare.
Publications
- Adenosine-A1 receptor agonist induced hyperalgesic priming type II.
- Antihyperalgesic effect of tetrodotoxin in rat models of persistent muscle pain.
- Variations in potassium channel genes are associated with distinct trajectories of persistent breast pain after breast cancer surgery.
- Neonatal handling (resilience) attenuates water-avoidance stress induced enhancement of chronic mechanical hyperalgesia in the rat.
- Plasma Membrane Mechanisms in a Preclinical Rat Model of Chronic Pain.
- Accounting for the delay in the transition from acute to chronic pain: axonal and nuclear mechanisms.
- Homocysteine-induced attenuation of vascular endothelium-dependent hyperalgesia in the rat.
- Preoperative Breast Pain Predicts Persistent Breast Pain and Disability Following Breast Cancer Surgery.
- Persistent arm pain is distinct from persistent breast pain following breast cancer surgery.
- Persistent breast pain following breast cancer surgery is associated with persistent sensory changes, pain interference, and functional impairments.
- Polymorphisms in Tumor Necrosis Factor-α Are Associated With Higher Anxiety Levels in Women After Breast Cancer Surgery.
- Role of Nociceptor Estrogen Receptor GPR30 in a Rat Model of Endometriosis Pain.
- Does the antihyperalgesic disruptor of endothelial cells, octoxynol-9, alter nociceptor function?
- Side of Cancer Does Not Influence Limb Volumes in Women Prior to Breast Cancer Surgery.
- Impact of surgical excision of lesions on pain in a rat model of endometriosis.
- ATP release mechanisms of endothelial cell-mediated stimulus-dependent hyperalgesia.
- Screening the Role of Pro-Nociceptive Molecules in a Rodent Model of Endometriosis Pain.
- Role for Monocyte Chemoattractant Protein-1 (MCP-1) in the Induction of Chronic Muscle Pain in the Rat.
- Second messengers mediating the expression of neuroplasticity in a model of chronic pain in the rat.
- Associations between cytokine gene variations and severe persistent breast pain in women following breast cancer surgery.
- Identification of patient subgroups and risk factors for persistent arm/shoulder pain following breast cancer surgery.
- Persistent postoperative pain and sensory changes following lymph node excision in melanoma patients: a topical review.
- The fundamental unit of pain is the cell.
- Neural mechanisms of pain and alcohol dependence.
- NOP receptor mediates anti-analgesia induced by agonist-antagonist opioids.
- Ectopic endometrium-derived leptin produces estrogen-dependent chronic pain in a rat model of endometriosis.
- Epigenetic regulation and measurement of epigenetic changes.
- Role of Nociceptor αCaMKII in Transition from Acute to Chronic Pain (Hyperalgesic Priming) in Male and Female Rats.
- Electrophysiological correlates of hyperalgesic priming in vitro and in vivo.
- Stress in the Adult Rat Exacerbates Muscle Pain Induced by Early-Life Stress.
- Peripheral Administration of Translation Inhibitors Reverses Increased Hyperalgesia in a Model of Chronic Pain in the Rat.
- Vascular Endothelial Cells Mediate Mechanical Stimulation-Induced Enhancement of Endothelin Hyperalgesia via Activation of P2X2/3 Receptors on Nociceptors.
- Role of a novel nociceptor autocrine mechanism in chronic pain.
- Pain facilitation brain regions activated by nalbuphine are revealed by pharmacological FMRI.
- Role of endothelial cells in antihyperalgesia induced by a triptan and ?-blocker.
- Identification of patient subgroups and risk factors for persistent breast pain following breast cancer surgery.
- In Vivo and in Vitro Comparison of Female and Male Nociceptors.
- Independent contributions of alcohol and stress axis hormones to painful peripheral neuropathy.
- IB4(+) nociceptors mediate persistent muscle pain induced by GDNF.
- Ectopic uterine tissue as a chronic pain generator.
- Transient decrease in nociceptor GRK2 expression produces long-term enhancement in inflammatory pain.
- GDNF induces mechanical hyperalgesia in muscle by reducing I(BK) in IB4+ nociceptors.
- Associations Between Pro- and Anti-Inflammatory Cytokine Genes and Breast Pain in Women Prior to Breast Cancer Surgery.
- Association between pro- and anti-inflammatory cytokine genes and a symptom cluster of pain, fatigue, sleep disturbance, and depression.
- IB4-saporin attenuates acute and eliminates chronic muscle pain in the rat.
- Sexual dimorphism in endothelin-1 induced mechanical hyperalgesia in the rat.
- Early-life stress produces muscle hyperalgesia and nociceptor sensitization in the adult rat.
- Role of drp1, a key mitochondrial fission protein, in neuropathic pain.
- Muscle pain in models of chemotherapy-induced and alcohol-induced peripheral neuropathy.
- Mitochondrial dependence of nerve growth factor-induced mechanical hyperalgesia.
- Abnormal Muscle Afferent Function in a Model of Taxol Chemotherapy-induced Painful Neuropathy.
- Estrogen destabilizes microtubules through an ion-conductivity-independent TRPV1 pathway.
- Further Validation of a Model of Fibromyalgia Syndrome in the Rat.
- Nucleus accumbens facilitates nociception.
- Enhanced cytokine-induced mechanical hyperalgesia in skeletal muscle produced by a novel mechanism in rats exposed to unpredictable sound stress.
- Pain and death: neurodegenerative disease mechanisms in the nociceptor.
- Attenuation of activity in an endogenous analgesia circuit by ongoing pain in the rat.
- Mechanisms mediating vibration-induced chronic musculoskeletal pain analyzed in the rat.
- Shared mechanisms for opioid tolerance and a transition to chronic pain.
- Sound stress-induced long-term enhancement of mechanical hyperalgesia in rats is maintained by sympathoadrenal catecholamines.
- TRPC1 and TRPC6 channels cooperate with TRPV4 to mediate mechanical hyperalgesia and nociceptor sensitization.
- Comparison of oxaliplatin- and cisplatin-induced painful peripheral neuropathy in the rat.
- Stress induces a switch of intracellular signaling in sensory neurons in a model of generalized pain.
- Muscle inflammation induces a protein kinase Cepsilon-dependent chronic-latent muscle pain.
- Oxaliplatin acts on IB4-positive nociceptors to induce an oxidative stress-dependent acute painful peripheral neuropathy.
- A subanalgesic dose of morphine eliminates nalbuphine anti-analgesia in postoperative pain.
- Interaction of transient receptor potential vanilloid 4, integrin, and SRC tyrosine kinase in mechanical hyperalgesia.
- Open-label exploration of an intravenous nalbuphine and naloxone mixture as an analgesic agent following gynecologic surgery.
- Signaling pathways in sensitization: toward a nociceptor cell biology.
- Burn injury pain: the continuing challenge.
- TrkA and PKC-epsilon in thermal burn-induced mechanical hyperalgesia in the rat.
- Neurogenic inflammation and arthritis.
- A transient receptor potential vanilloid 4-dependent mechanism of hyperalgesia is engaged by concerted action of inflammatory mediators.
- Neuroleptics antagonize nalbuphine antianalgesia.
- Epinephrine-induced excitation and sensitization of rat C-fiber nociceptors.
- Epac mediates a cAMP-to-PKC signaling in inflammatory pain: an isolectin B4(+) neuron-specific mechanism.
- Transient receptor potential vanilloid 4 is essential in chemotherapy-induced neuropathic pain in the rat.
- Role of the sensory neuron cytoskeleton in second messenger signaling for inflammatory pain.
- Hypotonicity induces TRPV4-mediated nociception in rat.
- Vagal modulation of bradykinin-induced mechanical hyperalgesia in the female rat.
- Gonadal hormones do not account for sexual dimorphism in vagal modulation of nociception in the rat.
- Adaptations in nucleus accumbens circuitry during opioid withdrawal associated with persistence of noxious stimulus-induced antinociception in the rat.
- National Institutes of Health Workshop: reflex sympathetic dystrophy/complex regional pain syndromes--state-of-the-science.
- Sympathetic-independent bradykinin mechanical hyperalgesia induced by subdiaphragmatic vagotomy in the rat.
- Altered nucleus accumbens circuitry mediates pain-induced antinociception in morphine-tolerant rats.
- Bradykinin-12-lipoxygenase-VR1 signaling pathway for inflammatory hyperalgesia.
- Contribution of Piezo2 to endothelium-dependent pain.
- Mechanisms mediating nitroglycerin-induced delayed-onset hyperalgesia in the rat.
- Gene Expression Profiling of Evening Fatigue in Women Undergoing Chemotherapy for Breast Cancer.
- Extracellular Matrix Hyaluronan Signals Via its CD44 Receptor in the Increased Responsiveness to Mechanical Stimulation.
- Comparison of subgroups of breast cancer patients on pain and co-occurring symptoms following chemotherapy.
- Co-occurrence of anxiety and depressive symptoms following breast cancer surgery and its impact on quality of life.
- Predictors of Altered Upper Extremity Function During the First Year After Breast Cancer Treatment.
Schools
University of California At San Francisco
Kaiser Hospital
Doctors Specialties
- Internal Medicine
- Rheumatology
Accepted Insurances
Awards
- Elected Member
- Fellow (FACR)
Education
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UCSF -
Kaiser Permanente Medical Center -
University of California San Francisco School of Medicine
Drug Facts
| NPI NUMBER | 1821025479 | |
| NPPES Provider LastName | LEVINE | |
| NPPES Provider FirstName | JON | |
| NPPES Provider ZIPCode | 941432206 | |
| NPPES Provider State | CA | |
| Specialty Description | Rheumatology | |
| Total Claim Count | 20.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1821025479 |
| Last Name Of The Provider | LEVINE |
| First Name Of The Provider | JON |
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