Associate Professor of Medicine, Johns Hopkins University School of Medicine
Cardiology
Dr. Lili A. Barouch, MD
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Lili Barouch is an expert in the field of Cardiology. He went to Johns Hopkins University School of Medicine. Johns Hopkins University School of Medicine is one of the top medical schools in the US. Johns Hopkins University School of Medicine is ranked 3 in research and 29 in primary care medicine He received awards:"CMS Meaningful Use Stage 1 Certification" and "Fellow (FACC)". Lili Barouch is a published doctor. He has 33 publications published. The most current publication "Prognostic significance of microvascular obstruction by magnetic resonance imaging in patients with acute myocardial infarction." He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Anti-hypertrophic and anti-oxidant effect of beta3-adrenergic stimulation in myocytes requires differential neuronal NOS phosphorylation.
- Beneficial Cardiac Effects of Caloric Restriction Are Lost with Age in a Murine Model of Obesity.
- Interleukin 10 knockout frail mice develop cardiac and vascular dysfunction with increased age.
- Beta3-adrenoreceptor stimulation ameliorates Myocardial ischemia-reperfusion injury via endothelial nitric oxide synthase and neuronal nitric oxide synthase activation.
- Exercise protects against Myocardial ischemia-reperfusion injury via stimulation of ?(3)-adrenergic receptors and increased nitric oxide signaling: role of nitrite and...
- Restoring leptin signaling reduces hyperlipidemia and improves vascular stiffness induced by Chronic intermittent hypoxia.
- Caloric Restriction in Leptin Deficiency Does Not Correct Myocardial Steatosis: Failure to Normalize PPAR{alpha}/PGC1{alpha} and Thermogenic Glyecrolipid/Fatty Acid Cy...
- β2-adrenergic receptor-coupled phosphoinositide 3-kinase constrains cAMP-dependent increases in cardiac inotropy through phosphodiesterase 4 activation.
- The change in B-type natriuretic peptide levels over time predicts significant rejection in cardiac transplant recipients.
- Resolution of giant cell myocarditis after extended ventricular assistance.
- Cardiomyocyte apoptosis in animal models of obesity.
- Decreased p110alpha catalytic activity accompanies increased myocyte apoptosis and cardiac hypertrophy in leptin deficient ob/ob mice.
- New clinical and ultrastructural findings in hydroxychloroquine-induced Cardiomyopathy--a report of 2 cases.
- Leptin signaling and obesity: cardiovascular consequences.
- Reduced neuronal nitric oxide synthase expression contributes to cardiac oxidative stress and nitroso-redox imbalance in ob/ob mice.
- Activation of the cardiac ciliary neurotrophic factor receptor reverses left ventricular hypertrophy in leptin-deficient and leptin-resistant obesity.
- Xanthine oxidoreductase inhibition causes reverse remodeling in rats with dilated Cardiomyopathy.
- Cardiac myocyte apoptosis is associated with increased DNA damage and decreased survival in murine models of obesity.
- Deficiency of neuronal nitric oxide synthase increases mortality and cardiac remodeling after Myocardial infarction: role of nitroso-redox equilibrium.
- Nitric oxide and oxidative stress in cardiovascular aging.
- Critical role for the alpha-1B adrenergic receptor at the sympathetic neuroeffector junction.
- Disruption of leptin signaling contributes to cardiac hypertrophy independently of body weight in mice.
- Deficiency of different nitric oxide synthase isoforms activates divergent transcriptional programs in cardiac hypertrophy.
- Leptin repletion restores depressed {beta}-adrenergic contractility in ob/ob mice independently of cardiac hypertrophy.
- Effect of intermittent hypoxia on atherosclerosis in apolipoprotein E-deficient mice.
- PDE5A suppression of Acute beta-adrenergic activation requires modulation of myocyte beta-3 signaling coupled to PKG-mediated troponin I phosphorylation.
- Beta 3-adrenoreceptor regulation of nitric oxide in the cardiovascular system.
- Combined loss of neuronal and endothelial nitric oxide synthase causes premature mortality and age-related hypertrophic cardiac remodeling in mice.
- Nitric oxide regulates the Heart by spatial confinement of nitric oxide synthase isoforms.
- Adverse ventricular remodeling and exacerbated NOS uncoupling from pressure-overload in mice lacking the beta3-adrenoreceptor.
- Leptin is essential in maintaining normal vascular compliance independent of body weight.
- Prognostic significance of microvascular obstruction by magnetic resonance imaging in patients with Acute myocardial infarction.
Schools
Johns Hopkins Univ Sch Of Med
Johns Hopkins Hospital
Procedures Preformed
- Ablation for Treatment of Cardiac Arrhythmias
- Cardiac Catheterization (incl. Coronary Angiography)
- Cardiac Imaging
- Cardiac MRI (Magnetic Resonance Imaging) of Heart or Chest
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Conditions Treated
- Angina and Acute Coronary Syndrome
- Aortic Ectasia
- Aortic Valve Disease
- Arrhythmias (incl. Atrial Fibrillation)
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Doctors Specialties
- Cardiology
Accepted Insurances
- Blue Card PPO
- Blue Choice Advantage
- Blue Choice Network POS
- Blue Preferred PPO
- CareFirst Maryland POS
- Cigna HMO
- Cigna PPO
- Great West Healthcare-Cigna PPO
- Open Access Plus
- Southern Health PPO
- Coventry Health Care of Delaware PPO
- First Health (Coventry Health Care) PPO
- Choice Care PPO
- MultiPlan PPO
- PHCS Network PPO
- Choice Plus POS
- Navigate HMO
- Options PPO
Awards
- CMS Meaningful Use Stage 1 Certification
- Fellow (FACC)
Education
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Johns Hopkins University -
Johns Hopkins University School of Medicine
Hospital
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Howard County General Hospital -
Johns Hopkins Bayview Medical Center -
Johns Hopkins Hospital
Drug Facts
| NPI NUMBER | 1326003831 | |
| NPPES Provider LastName | BAROUCH | |
| NPPES Provider FirstName | LILI | |
| NPPES Provider ZIPCode | 212242735 | |
| NPPES Provider State | MD | |
| Specialty Description | Cardiology | |
| Total Claim Count | 373.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1326003831 |
| Last Name Of The Provider | BAROUCH |
| First Name Of The Provider | LILI |
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