Associate Professor, Anesthesiology and Physiology, Medical College of Wisconsin
Anesthesiology
Dr. Matthias Riess, MD
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Matthias Riess practices Anesthesiology. He graduated from Medical College of Wisconsin Affiliated Hospitals. Dr. Matthias Riess is a published doctor and has 55 publications published. The lastest was: 'Prejunctional neuropeptide Y receptors in human kidney and atrium.'
Publications
- Non-invasive cardiac output monitoring during sinus surgery.
- Bundled postconditioning therapies improve hemodynamics and neurologic recovery after 17min of untreated cardiac arrest.
- Anaesthetic postconditioning at the initiation of CPR improves myocardial and mitochondrial function in a pig model of prolonged untreated ventricular fibrillation.
- Opioid-Induced Cardioprotection.
- Genetically Determined Mitochondrial Preservation and Cardioprotection against Myocardial Ischemia-Reperfusion Injury in a Consomic Rat Model.
- Intralipid: the new magic bullet in cardioprotection?
- Reduced Mitochondrial Ca2+ Loading and Improved Functional Recovery after Ischemia Reperfusion Injury in Old vs. Young Guinea Pig Hearts.
- Severe Vasospasm Mimics Hypotension After High-Dose Intrauterine Vasopressin.
- Adding ROS Quenchers to Cold K+ Cardioplegia Reduces Superoxide Emission During 2-Hour Global Cold Cardiac Ischemia.
- Comparison of cumulative planimetry versus manual dissection to assess experimental infarct size in isolated hearts.
- Body mass index: an illogical correlate of obesity.
- The rocky road from bench to bedside: beta-blockers and anesthetic postconditioning.
- Two millimeters per inch.
- KATP channel openers have opposite effects on mitochondrial respiration under different energetic conditions.
- Differential increase of mitochondrial matrix volume by sevoflurane in isolated cardiac mitochondria.
- Near-embolization of a rubber core from a propofol vial.
- Reverse electron flow-induced ROS production is attenuated by activation of mitochondrial Ca2+-sensitive K+ channels.
- Lead poisoning in an adult: lead mobilization by pregnancy?
- ROS scavenging before 27 degrees C ischemia protects hearts and reduces mitochondrial ROS, Ca2+ overload, and changes in redox state.
- beta-Blockade abolishes anesthetic preconditioning: impact on clinical applicability.
- Mitochondrial Ca2+-induced K+ influx increases respiration and enhances ROS production while maintaining membrane potential.
- Hemoglobin M (Milwaukee) affects arterial oxygen saturation and makes pulse oximetry unreliable.
- Ischemia-reperfusion injury changes the dynamics of Ca2+-contraction coupling due to inotropic drugs in isolated hearts.
- Cardiac mitochondrial preconditioning by Big Ca2+-sensitive K+ channel opening requires superoxide radical generation.
- Increasing heart size and age attenuate anesthetic preconditioning in guinea pig isolated hearts.
- Improved mitochondrial bioenergetics by anesthetic preconditioning during and after 2 hours of 27 degrees C ischemia in isolated hearts.
- Warm ischemic preconditioning improves mitochondrial redox balance during and after mild hypothermic ischemia in guinea pig isolated hearts.
- Anesthetic preconditioning: the role of free radicals in sevoflurane-induced attenuation of mitochondrial electron transport in Guinea pig isolated hearts.
- Negative inotropic drugs alter indexes of cytosolic [Ca(2+)]-left ventricular pressure relationships after ischemia.
- Cardiac pharmacological preconditioning with volatile anesthetics: from bench to bedside?
- Hypothermia augments reactive oxygen species detected in the guinea pig isolated perfused heart.
- Dual exposure to sevoflurane improves anesthetic preconditioning in intact hearts.
- Attenuation of mitochondrial respiration by sevoflurane in isolated cardiac mitochondria is mediated in part by reactive oxygen species.
- Reactive oxygen species and cardiac preconditioning: many questions remain.
- How inotropic drugs alter dynamic and static indices of cyclic myoplasmic [Ca2+] to contractility relationships in intact hearts.
- Anesthetic preconditioning: effects on latency to ischemic injury in isolated hearts.
- Sevoflurane exposure generates superoxide but leads to decreased superoxide during ischemia and reperfusion in isolated hearts.
- Ischemic preconditioning alters real-time measure of O2 radicals in intact hearts with ischemia and reperfusion.
- Preconditioning with sevoflurane reduces changes in nicotinamide adenine dinucleotide during ischemia-reperfusion in isolated hearts: reversal by 5-hydroxydecanoic acid.
- Anesthetic preconditioning attenuates mitochondrial Ca2+ overload during ischemia in Guinea pig intact hearts: reversal by 5-hydroxydecanoic acid.
- Sevoflurane preconditioning before moderate hypothermic ischemia protects against cytosolic [Ca(2+)] loading and myocardial damage in part via mitochondrial K(ATP) cha...
- Anesthetic preconditioning: triggering role of reactive oxygen and nitrogen species in isolated hearts.
- Altered NADH and improved function by anesthetic and ischemic preconditioning in guinea pig intact hearts.
- Cardiac preconditioning with 4-h, 17 degrees C ischemia reduces [Ca(2+)](i) load and damage in part via K(ATP) channel opening.
- Far red/near infrared light-induced protection against cardiac ischemia and reperfusion injury remains intact under diabetic conditions and is independent of nitric ox...
- Implementing a successful journal club in an anesthesiology residency program.
- Refractory hypotension during general anesthesia despite preoperative discontinuation of an angiotensin receptor blocker.
- Sedation assessment in critically ill patients with bispectral index.
- Prejunctional neuropeptide Y receptors in human kidney and atrium.
- Cardiotonic drugs differentially alter cytosolic [Ca2+] to left ventricular relationships before and after ischemia in isolated guinea pig hearts.
- Ischemia reperfusion dysfunction changes model-estimated kinetics of myofilament interaction due to inotropic drugs in isolated hearts.
- Bispectral Index monitoring in Intensive Care Units: are we really there yet?
- Comparison of hyperkalemic cardioplegia with altered [CaCl2] and [MgCl2] on [Ca2+]i transients and function after warm global ischemia in isolated hearts.
- Reduced reactive O2 species formation and preserved mitochondrial NADH and [Ca2+] levels during short-term 17 degrees C ischemia in intact hearts.
Doctors Specialties
- Anesthesiology
Accepted Insurances
Education
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Medical College of Wisconsin Affiliated Hospitals -
Albert Ludwigs University Faculty of Medicine
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1083796320 |
| Last Name Of The Provider | RIESS |
| First Name Of The Provider | MATTHIAS |
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