Physician
Neurology
Dr. Michel Toledano, MD
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Michel Toledano, MD works in the field of Neurology. Michel Toledano is a published doctor. He published 47 publications, including: 'A new antioxidant with alkyl hydroperoxide defense properties in yeast.' He accepts Medicare payments and is listed with Medicare.gov.
Publications
- Adaptive aneuploidy protects against thiol peroxidase deficiency by increasing respiration via key mitochondrial proteins.
- Teaching NeuroImages: diagnostic utility of FDG-PET in neurolymphomatosis.
- [Are mild ovarian stimulations for IVF-ET a significant progress in assisted reproductive technologies?].
- Dental composites reinforced with hydroxyapatite: mechanical behavior and absorption/elution characteristics.
- Hydrolytic stability of experimental hydroxyapatite-filled dental composite materials.
- Reversal of compromised bonding in bleached enamel.
- Reversal of compromised bonding to oxidized etched dentin.
- Spatially resolved photopolymerization kinetics and oxygen inhibition in dental adhesives.
- Interaction between iron metabolism and 2,3,7,8-tetrachlorodibenzo-p-dioxin in mice with variants of the Ahr gene: a hepatic oxidative mechanism.
- A new antioxidant with alkyl hydroperoxide defense properties in yeast.
- Yap1 and Skn7 control two specialized oxidative stress response regulons in yeast.
- A genetic investigation of the essential role of glutathione: mutations in the proline biosynthesis pathway are the only suppressors of glutathione auxotrophy in yeast.
- The H2O2 stimulon in Saccharomyces cerevisiae.
- A proteome analysis of the cadmium response in Saccharomyces cerevisiae.
- H2O2 sensing through oxidation of the Yap1 transcription factor.
- Yap8p activation in Saccharomyces cerevisiae under arsenic conditions.
- Ionizing radiation induces a Yap1-dependent peroxide stress response in yeast.
- Visualization of ribonucleotide reductase catalytic oxidation establishes thioredoxins as its major reductants in yeast.
- alpha1-Antitrypsin monotherapy induces immune tolerance during islet allograft transplantation in mice.
- Life span extension and H(2)O(2) resistance elicited by caloric restriction require the peroxiredoxin Tsa1 in Saccharomyces cerevisiae.
- Glutathione revisited: a vital function in iron metabolism and ancillary role in thiol-redox control.
- Nitric oxide activates an Nrf2/sulfiredoxin antioxidant pathway in macrophages.
- Sulfiredoxin protects mice from lipopolysaccharide-induced endotoxic shock.
- Proteome screens for Cys residues oxidation: the redoxome.
- Reining in H(2)O(2) for safe signaling.
- Activation of NRF2 by nitrosative agents and H2O2 involves KEAP1 disulfide formation.
- H2O2 activates the nuclear localization of Msn2 and Maf1 through thioredoxins in Saccharomyces cerevisiae.
- The guardian recruits cops: the p53-p21 axis delegates prosurvival duties to the Keap1-Nrf2 stress pathway.
- Dug1p Is a Cys-Gly peptidase of the gamma-glutamyl cycle of Saccharomyces cerevisiae and represents a novel family of Cys-Gly peptidases.
- Protein-thiol oxidation, from single proteins to proteome-wide analyses.
- The dual functions of thiol-based peroxidases in H2O2 scavenging and signaling.
- Regulation of peroxiredoxins by nitric oxide in immunostimulated macrophages.
- ROS as signalling molecules: mechanisms that generate specificity in ROS homeostasis.
- The system biology of thiol redox system in Escherichia coli and yeast: differential functions in oxidative stress, iron metabolism and DNA synthesis.
- Disulfide Bond-mediated multimerization of Ask1 and its reduction by thioredoxin-1 regulate H(2)O(2)-induced c-Jun NH(2)-terminal kinase activation and apoptosis.
- Cytoplasmic glutathione redox status determines survival upon exposure to the thiol-oxidant 4,4''-dipyridyl disulfide.
- The Arabidopsis thaliana sulfiredoxin is a plastidic cysteine-sulfinic acid reductase involved in the photooxidative stress response.
- The S. cerevisiae Yap1 and Yap2 transcription factors share a common cadmium-sensing domain.
- The Saccharomyces cerevisiae proteome of oxidized protein thiols: contrasted functions for the thioredoxin and glutathione pathways.
- A cysteine-sulfinic acid in peroxiredoxin regulates H2O2-sensing by the antioxidant Pap1 pathway.
- Microbial H2O2 sensors as archetypical redox signaling modules.
- Mammalian antioxidant defenses are not inducible by H2O2.
- ATP-dependent reduction of cysteine-sulphinic acid by S. cerevisiae sulphiredoxin.
- Two redox centers within Yap1 for H2O2 and thiol-reactive chemicals signaling.
- A thiol peroxidase is an H2O2 receptor and redox-transducer in gene activation.
- The control of the yeast H2O2 response by the Msn2/4 transcription factors.
Conditions Treated
- Convulsions
- Epilepsy
- Gait Abnormality
- Headache
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Doctors Specialties
- Neurology
Accepted Insurances
Education
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Mayo School of Graduate Medical Education
Hospital
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Mayo Clinic - Rochester, Minnesota
Drug Facts
| NPI NUMBER | 1649581166 | |
| NPPES Provider LastName | TOLEDANO | |
| NPPES Provider FirstName | MICHEL | |
| NPPES Provider ZIPCode | 559050001 | |
| NPPES Provider State | MN | |
| Specialty Description | Neurology | |
| Total Claim Count | 51.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1649581166 |
| Last Name Of The Provider | TOLEDANO |
| First Name Of The Provider | MICHEL |
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