Assistant Professor, Medical Genetics, Mayo Medical School
Medical Genetics
Dr. Pavel N. Pichurin, MD
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Doctor Pavel N. Pichurin, MD is an expert in the field of Medical Genetics. He studied medicine at University of Texas Southwestern Medical School. Pavel Pichurin is a published doctor. He has 35 papers published. The most current publication "Ehlers Danlos syndrome and gastrointestinal manifestations: a 20-year experience at Mayo Clinic." Dr. Pavel N. Pichurin accepts Medicare payments and is registered with Medicare.gov.
Publications
- Ehlers Danlos syndrome and gastrointestinal manifestations: a 20-year experience at Mayo Clinic.
- Appreciating the broad clinical features of SMAD4 mutation carriers: a multicenter chart review.
- Shared and unique susceptibility genes in a mouse model of Graves' disease determined in BXH and CXB recombinant inbred mice.
- The link between Graves' disease and Hashimoto's thyroiditis: a role for regulatory T cells.
- Probing the genetic basis for thyrotropin receptor antibodies and hyperthyroidism in immunized CXB recombinant inbred mice.
- Targeted expression of the human thyrotropin receptor A-subunit to the mouse thyroid: insight into overcoming the lack of response to A-subunit adenovirus immunization.
- Relationship between thyroid peroxidase T cell epitope restriction and antibody recognition of the autoantibody immunodominant region in human leukocyte antigen DR3 tr...
- Dissociation between iodide-induced thyroiditis and antibody-mediated hyperthyroidism in NOD.H-2h4 mice.
- "Hijacking" the thyrotropin receptor: A chimeric receptor-lysosome associated membrane protein enhances deoxyribonucleic acid vaccination and induces Graves' hyperthyr...
- Naked deoxyribonucleic acid vaccination induces recognition of diverse thyroid peroxidase T cell epitopes.
- Thyrotropin receptor knockout mice: studies on immunological tolerance to a major thyroid autoantigen.
- The cysteine-rich amino terminus of the thyrotropin receptor is the immunodominant linear antibody epitope in mice immunized using naked deoxyribonucleic acid or adeno...
- Evidence that the complement control protein-epidermal growth factor-like domain of thyroid peroxidase lies on the fringe of the immunodominant region recognized by au...
- Dendritic cells infected with adenovirus expressing the thyrotrophin receptor induce Graves'' hyperthyroidism in BALB/c mice.
- Thyroglobulin-thyroperoxidase autoantibodies are polyreactive, not bispecific: analysis using human monoclonal autoantibodies.
- Peptide scanning for thyrotropin receptor T-cell epitopes in mice vaccinated with naked DNA.
- Thyroid-stimulating autoantibodies in Graves disease preferentially recognize the free A subunit, not the thyrotropin holoreceptor.
- Immune deviation away from Th1 in interferon-gamma knockout mice does not enhance TSH receptor antibody production after naked DNA vaccination.
- Naked TSH receptor DNA vaccination: A TH1 T cell response in which interferon-gamma production, rather than antibody, dominates the immune response in mice.
- Human monoclonal autoantibodies to B-cell epitopes outside the thyroid peroxidase autoantibody immunodominant region.
- A prion-like shift between two conformational forms of a recombinant thyrotropin receptor A-subunit module: purification and stabilization using chemical chaperones of...
- Cytokines, IgG subclasses and costimulation in a mouse model of thyroid autoimmunity induced by injection of fibroblasts co-expressing MHC class II and thyroid autoant...
- A direct binding assay for thyrotropin receptor autoantibodies.
- Thyrotropin receptor-DNA vaccination of transgenic mice expressing HLA-DR3 or HLA-DQ6b.
- Low-dose immunization with adenovirus expressing the thyroid-stimulating hormone receptor A-subunit deviates the antibody response toward that of autoantibodies in hum...
- The thyrotropin receptor autoantigen in Graves disease is the culprit as well as the victim.
- Insight into antibody responses induced by plasmid or adenoviral vectors encoding thyroid peroxidase, a major thyroid autoantigen.
- Evidence that factors other than particular thyrotropin receptor T cell epitopes contribute to the development of hyperthyroidism in murine Graves'' disease.
- Thyrotrophin receptor-specific memory T cell responses require normal B cells in a murine model of Graves'' disease.
- Susceptibility rather than resistance to hyperthyroidism is dominant in a thyrotropin receptor adenovirus-induced animal model of Graves' disease as revealed by BALB/c...
- Affinity-enrichment of thyrotropin receptor autoantibodies from Graves' patients and normal individuals provides insight into their properties and possible origin from...
- Interactions between the mannose receptor and thyroid autoantigens.
- HLA-DR3 transgenic mice immunized with adenovirus encoding the thyrotropin receptor: T cell epitopes and functional analysis of the CD40 Graves' polymorphism.
- IFNγ production by NK cells from HLA-sensitized patients after in vitro exposure to allo-antigens.
Doctors Specialties
- Clinical Genetics
- Medical Genetics
Accepted Insurances
Education
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University of Texas Southwestern Medical School -
Cedars-Sinai Medical Center -
Ural State Medical Academy
Hospital
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Mayo Clinic - Rochester, Minnesota
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1568697829 |
| Last Name Of The Provider | PICHURIN |
| First Name Of The Provider | PAVEL |
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