Associate Professor, Pathology and Laboratory Medicine, Indiana University School of Medicine
Pathology
Dr. Raymond Konger, MD
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Raymond Konger practices Pathology. He researched medicine at Indiana University School of Medicine. Indiana University School of Medicine is ranked 45/47 in Research/PrimaryCare. Raymond Konger is a published physician. He published 32 publications, including: 'Growth regulation of primary human keratinocytes by prostaglandin E receptor EP2 and EP3 subtypes.' He is registered with Medicare and accepts Medicare payments.
Publications
- Topical photodynamic therapy induces systemic immunosuppression via generation of platelet-activating factor receptor ligands.
- Chemotherapeutic agents subvert tumor immunity by generating agonists of platelet-activating factor.
- Cigarette Smoke Exposure Inhibits Contact Hypersensitivity via the Generation of Platelet-Activating Factor Agonists.
- Spatiotemporal assessments of dermal hyperemia enable accurate prediction of experimental cutaneous carcinogenesis as well as chemopreventive activity.
- Topical application of a platelet activating factor receptor agonist suppresses phorbol ester-induced acute and chronic inflammation and has cancer chemopreventive act...
- Systemic Platelet-activating Factor Receptor Activation Augments Experimental Lung Tumor Growth and Metastasis.
- The EP3 receptor stimulates ceramide and diacylglycerol release and inhibits growth of primary keratinocytes.
- Immunolocalization of low-affinity prostaglandin E receptors, EP and EP, in adult human epidermis.
- Epidermal peroxisome proliferator-activated receptor gamma as a target for ultraviolet B radiation.
- Regulation of parathyroid hormone-related protein gene expression by epidermal growth factor-family ligands in primary human keratinocytes.
- Pertussis toxin-sensitive secretory phospholipase A2 expression and motility in activated primary human keratinocytes.
- Loss of the EP2 prostaglandin E2 receptor in immortalized human keratinocytes results in increased invasiveness and decreased paxillin expression.
- Coordinate expression of secretory phospholipase A(2) and cyclooxygenase-2 in activated human keratinocytes.
- Growth regulation of primary human keratinocytes by prostaglandin E receptor EP2 and EP3 subtypes.
- Epidermal growth factor induces terminal differentiation in human epidermoid carcinoma cells.
- Enhancement factor in low-coherence enhanced backscattering and its applications for characterizing experimental skin carcinogenesis.
- The peroxisome proliferator-activated receptor gamma system regulates ultraviolet B-induced prostaglandin e(2) production in human epidermal keratinocytes.
- Ultraviolet B radiation of human skin generates platelet-activating factor receptor agonists.
- The EP1 subtype of prostaglandin E2 receptor: role in keratinocyte differentiation and expression in non-melanoma skin cancer.
- Ultraviolet B radiation generated platelet-activating factor receptor agonist formation involves EGF-R-mediated reactive oxygen species.
- Oxidized glycerophosphocholines as biologically active mediators for ultraviolet radiation-mediated effects.
- UVB radiation-mediated inhibition of contact hypersensitivity reactions is dependent on the platelet-activating factor system.
- Augmentation of UVB radiation-mediated early gene expression by the epidermal platelet-activating factor receptor.
- Inhibition of epidermal growth factor receptor signalling reduces hypercalcaemia induced by human lung squamous-cell carcinoma in athymic mice.
- Deletion of prostaglandin E2 EP2 receptor protects against ultraviolet-induced carcinogenesis, but increases tumor aggressiveness.
- A new wrinkle on topical vitamin E and photo-inflammation: Mechanistic studies of a hydrophilic gamma-tocopherol derivative compared with alpha-tocopherol.
- Involvement of PPARgamma in oxidative stress-mediated prostaglandin E(2) production in SZ95 human sebaceous gland cells.
- Increased skin barrier disruption by sodium lauryl sulfate in mice expressing a constitutively active STAT6 in T cells.
- Prostaglandin E receptor EP1 suppresses breast cancer metastasis and is linked to survival differences and cancer disparities.
- Microsomal prostaglandin E2 synthase-1 in breast cancer: a potential target for therapy.
- Prostanoid receptor EP1 expression in breast cancer.
Schools
Indiana University School Of Medicine
Barnes Jewish Hospital
University Rochester
Doctors Specialties
- Clinical Pathology
- Pathology
Accepted Insurances
Education
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Washington University - Barnes-Jewish Hospital -
Indiana University School of Medicine
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1760433379 |
| Last Name Of The Provider | KONGER |
| First Name Of The Provider | RAYMOND |
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