Clinical Fellow in Pathology, Massachusetts General Hospital

Pathology

Dr. Ricard Masia practices Pathology. He went to Washington University School of Medicine in St. Louis. Ricard Masia, MD is a published MD. He published 19 publications, including: 'New onset idiosyncratic liver enzyme elevations with biological therapy in inflammatory bowel disease.' Ricard Masia accepts Medicare.

Publications

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• New onset idiosyncratic liver enzyme elevations with biological therapy in inflammatory bowel disease.
• The inward rectifier potassium channel Kir2.1 is expressed in mouse neutrophils from bone marrow and liver.
• Survival in ampullary cancer: potential role of different KRAS mutations.
• Gastrointestinal biopsy findings of autoimmune enteropathy: a review of 25 cases.
• Mycobacterium chelonae Bacteremia After First Dose of Infliximab for Ulcerative Colitis.
• Cord Colitis Syndrome: A Cause of Granulomatous Inflammation in the Upper and Lower Gastrointestinal Tract.
• Case records of the Massachusetts General Hospital. Case 16-2013. A 12-year-old girl with irritability, hypersomnia, and somatic symptoms.
• Pancreatic mucinous cystic neoplasm of the main pancreatic duct.
• Pathology quiz case 1. Diffuse large B-cell lymphoma (DLBCL).
• Differential structure of atrial and ventricular KATP: atrial KATP channels require SUR1.
• Functional clustering of mutations in the dimer interface of the nucleotide binding folds of the sulfonylurea receptor.
• Regulation of KATP channel expression and activity by the SUR1 nucleotide binding fold 1.
• Arrhythmia susceptibility and premature death in transgenic mice overexpressing both SUR1 and Kir6.2[DeltaN30,K185Q] in the heart.
• A mutation in the TMD0-L0 region of sulfonylurea receptor-1 (L225P) causes permanent neonatal diabetes mellitus (PNDM).
• An ATP-binding mutation (G334D) in KCNJ11 is associated with a sulfonylurea-insensitive form of developmental delay, epilepsy, and neonatal diabetes.
• Expression of ATP-insensitive KATP channels in pancreatic beta-cells underlies a spectrum of diabetic phenotypes.
• Transgenic overexpression of SUR1 in the heart suppresses sarcolemmal K(ATP).
• Differential nucleotide regulation of KATP channels by SUR1 and SUR2A.

Schools

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• Washington Center School of Medicine

Doctors Specialties

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• Anatomic Pathology
• Pathology

Accepted Insurances

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• GHI PPO
• First Health (Coventry Health Care) PPO
• Harvard Pilgrim Health Care PPO
• Humana
• Medicare MCR
• Choice Plus POS
• Navigate HMO
• Navigate POS
• Options PPO

Education

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• Massachusetts General Hospital
• Washington University School of Medicine in St. Louis

Hospital

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• Massachusetts General Hospital

Helpful Reviews

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Medicare Facts

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National Provider Identifier [NPI]	1780843110
Last Name Of The Provider	MASIA
First Name Of The Provider	RICARD
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