Professor, Neurology, UC, Davis School of Medicine
Neurology
Dr. Ricardo A. Maselli, MD
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Ricardo Maselli practices Neurology. He received 1 awards: "CMS Meaningful Use Stage 1 Certification". Ricardo Maselli is a published doctor. He has 75 publications published. The most current publication "Morvan''s fibrillary chorea: a paraneoplastic manifestation of thymoma." He accepts Medicare payments and is listed with Medicare.gov.
Publications
- Editorial by concerned physicians: Unintended effect of the orphan drug act on the potential cost of 3,4-diaminopyridine.
- Axonal neuropathy in female carriers of the fragile X premutation with fragile X-associated tremor Ataxia syndrome.
- COOH-terminal collagen Q (COLQ) mutants causing human deficiency of endplate acetylcholinesterase impair the interaction of ColQ with proteins of the basal lamina.
- Synaptic basal lamina-associated congenital myasthenic Syndromes.
- Dominantly Inherited Myotonia Congenita Resulting from a Mutation That Increases Open Probability of the Muscle Chloride Channel CLC-1.
- LG2 agrin mutation causing severe congenital myasthenic Syndrome mimics functional characteristics of non-neural (z-) agrin.
- Acute Severe Animal Model of Anti-Muscle-Specific Kinase Myasthenia: Combined Postsynaptic and Presynaptic Changes.
- Protection of human muscle acetylcholinesterase from soman by pyridostigmine bromide.
- Variable phenotypes associated with mutations in DOK7.
- High throughput genetic analysis of congenital myasthenic Syndromes using resequencing microarrays.
- Novel beta subunit mutation causes a slow-channel Syndrome by enhancing activation and decreasing the rate of agonist dissociation.
- Focal caspase activation underlies the endplate myopathy in slow-channel Syndrome.
- Rapsyn mutations in myasthenic Syndrome due to impaired receptor clustering.
- Effect of inherited abnormalities of calcium regulation on human neuroMuscular transmission.
- Choline acetyltransferase mutations in myasthenic Syndrome due to deficient acetylcholine resynthesis.
- Active calcium accumulation underlies severe weakness in a panel of mice with slow-channel Syndrome.
- Novel delta subunit mutation in slow-channel Syndrome causes severe weakness by novel mechanisms.
- Italian real-life experience of omalizumab.
- Retrospective analysis of adverse drug reactions to bronchodilators observed in two pulmonary divisions of Catanzaro, Italy.
- Adverse drug reactions to antibiotics observed in two pulmonology divisions of catanzaro, Italy: a six-year retrospective study.
- Increased activation of p38 MAPK in COPD.
- Experimental allergic neuritis in the SJL/J mouse: dysfunction of peripheral nerve without clinical signs.
- Intrafamilial heterogeneity in hereditary motor neuron Disease.
- Blood lymphocyte beta-adrenergic receptors in Multiple sclerosis.
- Pathophysiology of myasthenia gravis and Lambert-Eaton Syndrome.
- Sympathetic skin responses are decreased and lymphocyte beta-adrenergic receptors are increased in progressive Multiple sclerosis.
- Absence of F waves in heat stroke.
- Edrophonium: an aid in the diagnosis of acute organophosphate poisoning.
- Post-poliomyelitis Syndrome: evidence of ongoing denervation in symptomatic and asymptomatic patients.
- Late denervation in patients with antecedent paralytic poliomyelitis.
- Unsuspected V nerve lesion resulting from perineural cancer spread detected by blink reflex.
- Comparison of sympathetic skin response with quantitative sudomotor axon reflex test in Diabetic neuropathy.
- Hypertrophic peripheral Neuropathy due to leprosy: MR features.
- Inflammation at the neuroMuscular junction in myasthenia gravis.
- Diagnostic difficulties in patients with adul botulism type a.
- Effects of a monoclonal anti-acetylcholine receptor antibody on the avian end-plate.
- NeuroMuscular transmission as a function of motor unit size in patients with prior poliomyelitis.
- Single fiber electromyography of extraocular muscles: a sensitive method for the diagnosis of ocular Myasthenia gravis.
- In vitro microelectrode study of neuroMuscular transmission in a case of botulism.
- End-plate electromyography: use of spectral analysis of end-plate noise.
- Electrodiagnosis of Disorders of neuromuscular transmission.
- Pathogenesis of human botulism.
- Sympathetic skin response in Diabetic neuropathy.
- Single motor unit H-reflex in motor neuron Disorders.
- Analysis of the organophosphate-induced electromyographic response to repetitive nerve stimulation: paradoxical response to edrophonium and D-tubocurarine.
- Morvan''s fibrillary Chorea: a paraneoplastic manifestation of thymoma.
- Mutations in LAMB2 causing a severe form of synaptic congenital myasthenic Syndrome.
- Antibody effector mechanisms in Myasthenia gravis. The complement hypothesis.
- Effector mechanisms of myasthenic antibodies.
- Anconeus muscle: a human muscle preparation suitable for in-vitro microelectrode studies.
- AAEM case report 16. Botulism. American Association of Electrodiagnostic Medicine.
- Congenital myasthenic Syndrome caused by two non-N88K rapsyn mutations.
- Analysis of anticholinesterase-induced neuroMuscular transmission failure.
- Analysis of neuroMuscular transmission failure induced by anticholinesterases.
- Morvan's fibrillary Chorea. Electrodiagnostic and in vitro microelectrode findings.
- Antibodies to AChR, synapse-organizing proteins, titin, and other muscle proteins in Morvan's fibrillary Chorea.
- Rhodamine-labeled alpha-bungarotoxin allows visualization of end plates in congenital end plate acetylcholinesterase deficiency (CEAD).
- Fulminant demyelinating neuropathy mimicking Cerebral death.
- In vitro blockade of neuroMuscular transmission by monoclonal anti-acetylcholine receptor antibodies.
- Function and ultrastructure of the neuromuscular junction in post-polio Syndrome.
- Cluster of wound botulism in California: clinical, electrophysiologic, and pathologic study.
- Stimulated single-fiber electromyography in wound botulism.
- Neuromuscular transmission in amyotrophic lateral Sclerosis.
- Presynaptic failure of neuroMuscular transmission and synaptic remodeling in EA2.
- Presynaptic congenital myasthenic Syndrome due to quantal release deficiency.
- Genetic manipulation of AChR responses suggests multiple causes of weakness in slow-channel Syndrome.
- Slow-channel transgenic mice: a model of postsynaptic organellar degeneration at the neuroMuscular junction.
- A beta-subunit mutation in the acetylcholine receptor channel gate causes severe slow-channel Syndrome.
- Neuropathy as a presenting feature in fragile X-associated tremor/Ataxia syndrome.
- AbNormal nerve conduction features in fragile X premutation carriers.
- Evaluation of jitter by stimulated single-fiber electromyography in Normal dogs.
- Identification of pathogenic mutations in the human rapsyn gene.
- Common founder effect of rapsyn N88K studied using intragenic markers.
- Mutations in MUSK causing congenital myasthenic Syndrome impair MuSK-Dok-7 interaction.
Schools
University of Buenos Aires Faculty of Medicine
Thomason Genl Hospital
Italian Hospital
Kingston Genl Hospital Queens University
University of Chicago
Procedures Preformed
- EEG (Electroencephalogram)
- EMG (Electromyography)
Conditions Treated
- Alzheimer's Disease
- Ataxia
- Autonomic Disorders
- Bell's Palsy
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Doctors Specialties
- Neurology
Accepted Insurances
Awards
- CMS Meaningful Use Stage 1 Certification
Education
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University of Chicago -
University of Buenos Aires
Hospital
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University of California, Davis Medical Center
Drug Facts
| NPI NUMBER | 1972570638 | |
| NPPES Provider LastName | MASELLI | |
| NPPES Provider FirstName | RICARDO | |
| NPPES Provider ZIPCode | 958172201 | |
| NPPES Provider State | CA | |
| Specialty Description | Neurology | |
| Total Claim Count | 14.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1972570638 |
| Last Name Of The Provider | MASELLI |
| First Name Of The Provider | RICARDO |
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