Les Turner ALS Foundation/Herbert C. Wenske Foundation Professor, Northwestern University Medical School
Neurology
Dr. Teepu Siddique, MD
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Doctor Teepu Siddique, MD is an expert in the field of Neurology. He went to Dow Medical College. He over the years received 6 awards: "America's Top Doctors", "Chicago Magazine: Top Doctors", "Meaningful Use Stage 1 Certification", "The Forbes Norris Award", "Hope Through Caring Award" and "Sheila Essey Award". Dr. Teepu Siddique is a published physician. He has 61 papers published. The most current publication "Ophthalmic Manifestations of Amyotrophic Lateral Sclerosis (An American Ophthalmological Society Thesis)." He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Ophthalmic Manifestations of Amyotrophic Lateral Sclerosis (An American Ophthalmological Society Thesis).
- Sporadic and hereditary amyotrophic lateral Sclerosis (ALS).
- Mutation in the novel nuclear-encoded mitochondrial protein CHCHD10 in a family with autosomal dominant mitochondrial myopathy.
- Effect of fluoxetine on Disease progression in a mouse model of ALS.
- Discovery of 1,3,4-oxidiazole scaffold compounds as inhibitors of superoxide dismutase expression.
- Alzheimer Disease and amyotrophic lateral sclerosis: an etiopathogenic connection.
- inclusions in frontotemporal lobar degeneration with TDP-43 proteinopathy (FTLD-TDP) and amyotrophic lateral sclerosis (ALS), but not FTLD with FUS proteinopathy (FTLD...
- Prevalence and characteristics of Pain in early and late stages of ALS.
- Age and founder effect of SOD1 A4V mutation causing ALS.
- Protein recycling pathways in neurodegenerative Diseases.
- MspI RFLP for microtubule associated protein-2 (MAP2).
- UBQLN2/P62 cellular recycling pathways in amyotrophic lateral sclerosis and frontotemporal Dementia.
- SIGMAR1 mutations, genetic heterogeneity at the chromosome 9p locus, and the expanding etiological diversity of amyotrophic lateral Sclerosis.
- What is repeated in ALS and FTLD.
- SQSTM1 Mutations in Familial and Sporadic Amyotrophic Lateral Sclerosis.
- Detection of protein aggregation in neurodegenerative Diseases.
- Making connections: pathology and genetics link amyotrophic lateral sclerosis with frontotemporal lobe Dementia.
- Mutations in UBQLN2 cause dominant X-linked juvenile and adult-onset ALS and ALS/Dementia.
- Differential involvement of optineurin in amyotrophic lateral Sclerosis with or without SOD1 mutations.
- Confirmation of the severe phenotypic effect of serine at codon 41 of the superoxide dismutase 1 gene.
- Mutant TRPV4-mediated Toxicity is linked to increased constitutive function in axonal neuropathies.
- Recent advances in the genetics of hereditary axonal sensory-motor neuropathies type 2.
- An unusual case of familial ALS and cerebellar Ataxia.
- FUS-immunoreactive inclusions are a common feature in sporadic and non-SOD1 familial amyotrophic lateral sclerosis.
- Scapuloperoneal spinal muscular atrophy and CMT2C are allelic Disorders caused by alterations in TRPV4.
- Progressive changes in synaptic inputs to motoneurons in adult sacral spinal cord of a mouse model of amyotrophic lateral Sclerosis.
- Hyperactive intracellular calcium signaling associated with localized mitochondrial defects in skeletal muscle of an animal model of amyotrophic lateral Sclerosis.
- Marked accumulation of 27-hydroxycholesterol in SPG5 patients with hereditary spastic paresis.
- Wild-type SOD1 overexpression accelerates Disease onset of a G85R SOD1 mouse.
- Oculomotor involvement in myotonic dystrophy type 2.
- Genetics of amyotrophic lateral Sclerosis.
- Gender difference in levels of Cu/Zn superoxide dismutase (SOD1) in cerebrospinal fluid of patients with amyotrophic lateral Sclerosis.
- Molecular dissection of ALS-associated Toxicity of SOD1 in transgenic mice using an exon-fusion approach.
- What is missing in ALS.
- Sequence alterations within CYP7B1 implicate defective cholesterol homeostasis in motor-neuron degeneration.
- Restricted expression of mutant SOD1 in Spinal motor neurons and interneurons induces motor neuron pathology.
- Distal axonopathy in an alsin-deficient mouse model.
- Riluzole metabolism and CYP1A1/2 polymorphisms in patients with ALS.
- Gene expression analysis of frontotemporal lobar degeneration of the motor neuron Disease type with ubiquitinated inclusions.
- Pathological TDP-43 distinguishes sporadic amyotrophic lateral Sclerosis from amyotrophic lateral Sclerosis with SOD1 mutations.
- Genetic studies of autosomal recessive primary microcephaly in 33 Pakistani families: Novel sequence variants in ASPM gene.
- Conversion to the amyotrophic lateral Sclerosis phenotype is associated with intermolecular linked insoluble aggregates of SOD1 in mitochondria.
- Disulfide cross-linked protein represents a significant fraction of ALS-associated Cu, Zn-superoxide dismutase aggregates in Spinal cords of model mice.
- Informatics-assisted protein profiling in a transgenic mouse model of amyotrophic lateral Sclerosis.
- Familial amyotrophic lateral sclerosis with frontotemporal Dementia is linked to a locus on chromosome 9p13.2-21.3.
- Prion protein codon 129 genotype prevalence is altered in primary progressive aphasia.
- Apolipoprotein E is associated with age at onset of amyotrophic lateral Sclerosis.
- Genome-wide analysis of the parkinsonism-Dementia complex of Guam.
- Neuronal ubiquitinated intranuclear inclusions in familial and non-familial frontotemporal dementia of the motor neuron Disease type associated with amyotrophic latera...
- Presence of dendritic cells, MCP-1, and activated microglia/macrophages in amyotrophic lateral Sclerosis spinal cord tissue.
- Hyperexcitability of cultured Spinal motoneurons from presymptomatic ALS mice.
- A rare Cu/Zn superoxide dismutase mutation causing familial amyotrophic lateral sclerosis with variable age of onset, incomplete penetrance and a sensory Neuropathy.
- Phenotypic features and genetic findings in sacsin-related autosomal recessive Ataxia in Tunisia.
- Identification of two novel loci for dominantly inherited familial amyotrophic lateral Sclerosis.
- Two families with familial amyotrophic lateral Sclerosis are linked to a novel locus on chromosome 16q.
- A4T mutation in the SOD1 gene causing familial amyotrophic lateral Sclerosis.
- Cholinergic denervation in a pure multi-infarct state: observations on CADASIL.
- CuZn-superoxide dismutase in D90A heterozygotes from recessive and dominant ALS pedigrees.
- Frameshift mutation in the cartilage-derived morphogenetic protein 1 (CDMP1) gene and severe acromesomelic chondrodysplasia resembling Grebe-type chondrodysplasia.
- Genetic aspects of amyotrophic lateral Sclerosis.
Schools
Dow Medical College Dow University Of Health Sciences
Perth Amboy Genl Hospital
UMDNJ Nj Med School
Hospital For Spl Surgery Cornell Mc
National Institute Health
Procedures Preformed
- EEG (Electroencephalogram)
- EMG (Electromyography)
Conditions Treated
- Alzheimer's Disease
- Anterior Horn Disease
- Ataxia
- Bell's Palsy
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Doctors Specialties
- Neurology
Accepted Insurances
Awards
- America's Top Doctors
- Chicago Magazine: Top Doctors
- Meaningful Use Stage 1 Certification
- The Forbes Norris Award
- Hope Through Caring Award
- Sheila Essey Award
Education
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Hospital for Special Surgery -
UMDNJ-University Hospital -
Dow Medical College
Hospital
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Northwestern Memorial Hospital
Drug Facts
| NPI NUMBER | 1639187214 | |
| NPPES Provider LastName | SIDDIQUE | |
| NPPES Provider FirstName | TEEPU | |
| NPPES Provider ZIPCode | 606114546 | |
| NPPES Provider State | IL | |
| Specialty Description | Neurology | |
| Total Claim Count | 232.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1639187214 |
| Last Name Of The Provider | SIDDIQUE |
| First Name Of The Provider | TEEPU |
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