Professor, Radiation Oncology, Brown University Program in Medicine
Radiation Oncology
Dr. Timothy J. Kinsella, MD
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Doctor Timothy J. Kinsella, MD practices Radiation Oncology. He attended University of Rochester School of Medicine and Dentistry. He over the years received 6 awards: "Fellow", "Fellow, American Society of Therapeutic Radiology and Oncology", "Director's Service Award", "Master of Arts,ad eundem", "Award of Merit" and "Special Achievement Award". Timothy Kinsella has written publications. He has 55 publications published with PubMed. The lastest paper is: 'Updates in Molecular Pathology of Central Nervous System Gliomas in Adults.' Timothy J. Kinsella, MD accepts Medicare.
Publications
- Developing an in silico model of the modulation of base excision repair using methoxyamine for more targeted Cancer therapeutics.
- Analysis of cell cycle dynamics using probabilistic cell cycle models.
- Ribonucleotide reductase inhibition enhances chemoradiosensitivity of human cervical Cancers.
- Phase I trial of pelvic radiation, weekly cisplatin, and 3-aminopyridine-2-carboxaldehyde thiosemicarbazone (3-AP, NSC #663249) for locally advanced cervical Cancer.
- Postresection CA 19-9 value as a prognostic factor: in response to the letter by Kilickap and Arslan.
- Modulating radiation resistance by inhibiting ribonucleotide reductase in Cancers with virally or mutationally silenced p53 protein.
- Differential cellular responses to prolonged LDR-IR in MLH1-proficient and MLH1-deficient coloRectal cancer HCT116 cells.
- Essential role of DNA base excision repair on survival in an acidic tumor microenvironment.
- Outcomes of chemoradiotherapy with 5-Fluorouracil and mitomycin C for Anal cancer in immunocompetent versus immunodeficient patients.
- Modulation of the activity of methyl binding domain protein 4 (MBD4/MED1) while processing iododeoxyuridine generated DNA mispairs.
- Understanding DNA damage response and DNA repair pathways: applications to more targeted Cancer therapeutics.
- Coordination of DNA mismatch repair and base excision repair processing of chemotherapy and radiation damage for targeting resistant Cancers.
- Update on radiosensitization by halogenated thymidine Analogs-molecular mechanisms of drug processing and cell death signaling: implications for future clinical trials.
- The impact of resection margin status and postoperative CA19-9 levels on survival and patterns of recurrence after postoperative high-dose radiotherapy with 5-FU-based...
- Extraosseous Ewing's Sarcoma: 25 years later.
- Comparison of helical tomotherapy versus conventional radiation to deLiver craniospinal radiation.
- Mammalian target of rapamycin and S6 kinase 1 positively regulate 6-thioguanine-induced autophagy.
- Clinical results of intraoperative radiation therapy for patients with locally recurrent and advanced tumors having coloRectal involvement.
- Early experience with intraoperative radiotherapy in patients with resected pancreatic adenoCarcinoma.
- Full-dose gemcitabine with concurrent radiation therapy in patients with nonmetastatic pancreatic Cancer: a multicenter phase II trial.
- Guidelines for the use and interpretation of assays for monitoring autophagy in higher eukaryotes.
- Probabilistic modeling of DNA mismatch repair effects on cell cycle dynamics and iododeoxyuridine-DNA incorporation.
- 5-iodo-2-pyrimidinone-2'-deoxyribose-mediated cytotoxicity and radiosensitization in U87 human glioblastoma xenografts.
- Adjuvant therapy for Rectal cancer.
- BRCA1 activates a G2-M cell cycle checkpoint following 6-thioguanine-induced DNA mismatch damage.
- A novel role for DNA mismatch repair and the autophagic processing of chemotherapy drugs in human tumor cells.
- DNA mismatch repair initiates 6-thioguanine--induced autophagy through p53 activation in human tumor cells.
- The DNA N-glycosylase MED1 exhibits preference for halogenated pyrimidines and is involved in the cytotoxicity of 5-iododeoxyuridine.
- Methoxyamine potentiates iododeoxyuridine-induced radiosensitization by altering cell cycle kinetics and enhancing senescence.
- A multi-institutional phase II trial of preoperative full-dose gemcitabine and concurrent radiation for patients with potentially resectable pancreatic Carcinoma.
- The interaction between two radiosensitizers: 5-iododeoxyuridine and caffeine.
- Schedule-dependent drug effects of oral 5-iodo-2-pyrimidinone-2'-deoxyribose as an in vivo radiosensitizer in U251 human glioblastoma xenografts.
- CK2 inhibits apoptosis and changes its cellular localization following ionizing radiation.
- Casein kinase 2 regulates both apoptosis and the cell cycle following DNA damage induced by 6-thioguanine.
- Regularization of inverse planning for intensity-modulated radiotherapy.
- New radiosensitizing regimens, drugs, prodrugs, and candidates.
- Differential radiosensitization in DNA mismatch repair-proficient and -deficient human colon Cancer xenografts with 5-iodo-2-pyrimidinone-2'-deoxyribose.
- CHK1 and CHK2 are differentially involved in mismatch repair-mediated 6-thioguanine-induced cell cycle checkpoint responses.
- Continuous 28-day iododeoxyuridine infusion and hyperfractionated accelerated radiotherapy for Malignant glioma: a phase I clinical study.
- Mismatch repair-mediated G2/M arrest by 6-thioguanine involves the ATR-Chk1 pathway.
- Migration of implanted free radioactive seeds for adenoCarcinoma of the prostate using a Mick applicator.
- Real-time inverse planning for Gamma Knife radiosurgery.
- Intraoperative radiotherapy in the multimodality approach to bone and soft tissue Cancers.
- Normal Tissue tolerance to intraoperative radiotherapy.
- Four-year biochemical outcome after radioimmunoguided transperineal brachytherapy for patients with prostate adenoCarcinoma.
- Role of MutSalpha in the recognition of iododeoxyuridine in DNA.
- Overexpression of the R2 subunit of ribonucleotide reductase in human nasopharyngeal Cancer cells reduces radiosensitivity.
- Both DNA topoisomerase II-binding protein 1 and BRCA1 regulate the G2-M cell cycle checkpoint.
- DNA mismatch repair (MMR) mediates 6-thioguanine genotoxicity by introducing single-strand breaks to signal a G2-M arrest in MMR-proficient RKO cells.
- Synthesis and functional Analyses of nuclear clusterin, a cell death protein.
- Inhibition of base excision repair potentiates iododeoxyuridine-induced cytotoxicity and radiosensitization.
- Using intraoperative radiation therapy--a case study.
- A Predictive Genetic Signature for Response to Fluoropyrimidine-Based Neoadjuvant Chemoradiation in Clinical Stage II and III Rectal Cancer.
- Updates in Molecular Pathology of Central Nervous System Gliomas in Adults.
Schools
University Of Rochester
Mayo Clinic Rochester
Joint Center For Rad Therapy
Dana Farber Cancer Center
Procedures Preformed
- Stereotactic Radiosurgery, Spinal
Conditions Treated
- Acute Leukemia
- Anal and Rectal Cancer
- Anemia
- Bladder Cancer
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Doctors Specialties
- Medical Oncology
- Oncology
- Radiation Oncology
Accepted Insurances
Awards
- Fellow
- Fellow, American Society of Therapeutic Radiology and Oncology
- Director's Service Award
- Master of Arts,ad eundem
- Award of Merit
- Special Achievement Award
Education
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Mayo School of Graduate Medical Education -
University of Rochester School of Medicine and Dentistry
Hospital
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Rhode Island Hospital
Drug Facts
| NPI NUMBER | 1699709139 | |
| NPPES Provider LastName | KINSELLA | |
| NPPES Provider FirstName | TIMOTHY | |
| NPPES Provider ZIPCode | 029034801 | |
| NPPES Provider State | RI | |
| Specialty Description | Radiation Oncology | |
| Total Claim Count | 103.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1699709139 |
| Last Name Of The Provider | KINSELLA |
| First Name Of The Provider | TIMOTHY |
| View All |
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