Associate Professor of Surgery
General Surgery
Dr. Timothy Pritts, MD
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Doctor Timothy Pritts, MD works in the field of General Surgery. He graduated from Northwestern University Feinberg School of Medicine. Northwestern University Feinberg School of Medicine is ranked 19 in research and 29 in primary care medicine He has 3 awards "Regional Top Doctor", "CMS Meaningful Use Stage 1 Certification" and "Fellow (FACS)". Timothy Pritts is a published doctor. He has 65 papers published. The most current publication "Nuclear factor-kappaB is activated in intestinal mucosa during endotoxemia." He accepts Medicare payments and is registered with Medicare.gov.
Publications
- Prehospital airway technique does not influence incidence of ventilator-associated pneumonia in trauma patients.
- Macrophage-Derived Chemokine (CCL22) Is a Novel Mediator of Lung Inflammation Following Hemorrhage and Resuscitation.
- Preinjury alcohol exposure attenuates the neuroinflammatory response to traumatic brain injury.
- IL-6 Mediates Neuroinflammation and Motor Coordination Deficits After Mild Traumatic Brain Injury and Brief Hypoxia in Mice.
- Students' expectations on the surgery clerkship exceed those of residents and faculty.
- Implementation of a military-derived damage-control resuscitation strategy in a civilian trauma center decreases acute hypoxia in massively transfused patients.
- Application of the Berlin definition in PROMMTT patients: the impact of resuscitation on the incidence of hypoxemia.
- Human beta-defensin 3: a novel inhibitor of Staphylococcus-Produced biofilm production. Commentary on "Human β-defensin 3 inhibits antibiotic-resistant Staphylococcus ...
- A murine model of mild traumatic brain injury exhibiting cognitive and motor deficits.
- Platelets and Kupffer cells: partners in liver regeneration: Commentary on "Human platelets promote liver regeneration with Kupffer cells in SCID mice"
- Linalool: a novel natural anti-inflammatory agent?: Commentary on "Anti-inflammatory effects of linalool in RAW 264.7 macrophages and lipopolysaccharide-induced lung i...
- Sepsis-induced muscle proteolysis is prevented by a proteasome inhibitor in vivo.
- Proteasome inhibitors activate the transcription factors C/EBP-beta and delta in human intestinal epithelial cells.
- Expression and binding activity of the glucocorticoid receptor are upregulated in septic muscle.
- IL-1beta activates C/EBP-beta and delta in human enterocytes through a mitogen-activated protein kinase signaling pathway.
- Proteasome inhibitors induce heat shock response and increase IL-6 expression in human intestinal epithelial cells.
- Health-related quality of life after ileal pouch anal anastomosis for ulcerative colitis: right answer-wrong question.
- Management of common postoperative emergencies: are July interns ready for prime time?
- The University of Cincinnati College of Medicine Department of Surgery.
- Laparoscopic excision of a ciliated hepatic foregut cyst.
- Intestinal ischemia-reperfusion injury: reversible and irreversible damage imaged in vivo.
- Track configuration determines risk of driver injury in open wheel motor racing.
- Traumatic diaphragmatic injury: experience from a level I trauma center.
- Traumatic brain injury and aeromedical evacuation: when is the brain fit to fly?
- Remote thermal injury increases LPS-induced intestinal IL-6 production.
- Murine blood banking: characterization and comparisons to human blood.
- Omission of routine chest x-ray after chest tube removal is safe in selected trauma patients.
- Resuscitation with fresh whole blood ameliorates the inflammatory response after hemorrhagic shock.
- Development of a novel method of progressive temporary abdominal closure.
- Comparison of outcomes after laparoscopic versus open appendectomy for acute appendicitis at 222 ACS NSQIP hospitals.
- TNF-? induces vectorial secretion of IL-8 in Caco-2 cells.
- Hypobaric hypoxia exacerbates the neuroinflammatory response to traumatic brain injury.
- Hemorrhagic shock induces a proinflammatory milieu in the gut lumen.
- Ischemic post-conditioning to counteract intestinal ischemia/reperfusion injury.
- Proinflammatory chemokines in the intestinal lumen contribute to intestinal dysfunction during endotoxemia.
- The stress response decreases NF-kappaB activation in liver of endotoxemic mice.
- Transcription factors C/EBP-beta and -delta regulate IL-6 production in IL-1beta-stimulated human enterocytes.
- Managing morbid obesity: new options improve outcomes.
- Role of heat shock protein 70 in hepatic ischemia-reperfusion injury in mice.
- Cytokine-induced epithelial permeability changes are regulated by the activation of the p38 mitogen-activated protein kinase pathway in cultured Caco-2 cells.
- Learning styles vary among general surgery residents: analysis of 12 years of data.
- Gammadelta T cells mitigate the organ injury and mortality of sepsis.
- Activation of peroxisome proliferator-activated receptor-gamma during hepatic ischemia is age-dependent.
- The gene expression of ubiquitin ligase E3alpha is upregulated in skeletal muscle during sepsis in rats-potential role of glucocorticoids.
- IL-1beta induction of NF-kappaB activation in human intestinal epithelial cells is independent of oxyradical signaling.
- The devil is in the details: maximizing revenue for daily trauma care.
- Delayed presentation of thoracic esophageal perforation after blunt trauma.
- Quality of life in patients undergoing ileal pouch-anal anastomosis at the University of Cincinnati.
- Mucosal and enterocyte IL-6 production during sepsis and endotoxemia--role of transcription factors and regulation by the stress response.
- Nuclear factor-kappaB is activated in intestinal mucosa during endotoxemia.
- Complement component C3 production in IL-1beta-stimulated human intestinal epithelial cells is blocked by NF-kappaB inhibitors and by transfection with ser 32/36 mutan...
- Stress response decreases the interleukin-1beta-induced production of complement component C3 in human intestinal epithelial cells.
- Use of omeprazole in the management of giant duodenal ulcer: results of a prospective study.
- Implementation of a clinical pathway decreases length of stay and cost for bowel resection.
- Interleukin-1beta induces complement component C3 and IL-6 production at the basolateral and apical membranes in a human intestinal epithelial cell line.
- Induction of the stress response in vivo decreases nuclear factor-kappa B activity in jejunal mucosa of endotoxemic mice.
- Activation of NF-kappaB varies in different regions of the gastrointestinal tract during endotoxemia.
- The transcription factor activator protein-1 is activated and interleukin-6 production is increased in interleukin-1beta-stimulated human enterocytes.
- Induction of the stress response increases interleukin-6 production in the intestinal mucosa of endotoxaemic mice.
- Burn injuries in rats upregulate the gene expression of the ubiquitin-conjugating enzyme E2(14k) in skeletal muscle.
- Dantrolene reduces serum TNFalpha and corticosterone levels and muscle calcium, calpain gene expression, and protein breakdown in septic rats.
- Evaluation of endoscopic and laparoscopic training practices in surgical residency programs.
- Intraoperative manometry to assess the esophagogastric junction during laparoscopic fundoplication and myotomy.
- How residents learn predicts success in surgical residency.
Schools
Northwestern University The Feinberg School Of Medicine
University HospU Cincinnati
University Cincinnati
Doctors Specialties
- Critical Care Surgery
- General Surgery
Accepted Insurances
Awards
- Regional Top Doctor
- CMS Meaningful Use Stage 1 Certification
- Fellow (FACS)
Education
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University Hospital - University of Cincinnati College of Medicine -
Northwestern University Feinberg School of Medicine
Hospital
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Christ Hospital -
University of Cincinnati Medical Center
Drug Facts
| NPI NUMBER | 1194793786 | |
| NPPES Provider LastName | PRITTS | |
| NPPES Provider FirstName | TIMOTHY | |
| NPPES Provider ZIPCode | 452194231 | |
| NPPES Provider State | OH | |
| Specialty Description | General Surgery | |
| Total Claim Count | 30.0 | |
| Distinct Opioid Count | 0.0 | |
| Opioid Claim Count | 0.0 | |
| Percent Opioid Claims | 0.0 |
Helpful Reviews
Medicare Facts
| National Provider Identifier [NPI] | 1194793786 |
| Last Name Of The Provider | PRITTS |
| First Name Of The Provider | TIMOTHY |
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