Assistant Professor, Pediatrics, Ohio State University College of Medicine

Neonat/Perinatology

Trent Tipple practices Neonat/Perinatology. He researched medicine at Indiana University School of Medicine. Indiana University School of Medicine has a rank of 45 in research and a 47 in primary care He received awards:"Cassady Presidential Young Investigator Award", "Recognition Award for Outstanding Early Career Investigators", "Young Investigator Award" and "Resident research award". Trent Tipple has written publications. He has 16 publications published with PubMed. The lastest publication is: 'Arginase II is a target of miR-17-5p and regulates miR-17-5p expression in human pulmonary artery smooth muscle cells.'

Publications

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• Arginase II is a target of miR-17-5p and regulates miR-17-5p expression in human pulmonary artery smooth muscle cells.
• The Thioredoxin Reductase-1 Inhibitor Aurothioglucose Attenuates Lung Injury and Improves Survival in a Murine Model of Acute Respiratory Distress Syndrome.
• The Thioredoxin System in Neonatal Lung Disease.
• Thioredoxin-1 mediates hypoxia-induced pulmonary artery smooth muscle cell proliferation.
• Thioredoxin Reductase Inhibition Elicits Nrf2-mediated Responses in Clara Cells: Implications for Oxidant-induced Lung Injury.
• Plasma Lipid Metabolites are Associated with Gestational Age but Not Bronchopulmonary Dysplasia.
• Lipopolysaccharide-induced Cyclooxygenase-2 Expression in Mouse Transformed Clara Cells.
• Maternal docosahexaenoic acid supplementation decreases lung inflammation in hyperoxia-exposed newborn mice.
• Thioredoxin-interacting protein inhibits hypoxia-inducible factor transcriptional activity.
• Hyperoxia exposure alters hepatic eicosanoid metabolism in newborn mice.
• Alterations of the thioredoxin system by hyperoxia: implications for alveolar development.
• Differential responses in the lungs of newborn mouse pups exposed to 85% or >95% oxygen.
• Thioredoxin-related mechanisms in hyperoxic lung injury in mice.
• Riboflavin supplementation does not attenuate hyperoxic lung injury in transgenic (spc-mt)hGR mice.
• Neonatal hyperoxic exposure persistently alters lung secretoglobins and annexin A1.

Schools

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• Indiana University School Of Medicine

• Columbus Children39s Hospital

Doctors Specialties

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• Neonatal Medicine
• Neonat/Perinatology

Accepted Insurances

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Awards

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• Cassady Presidential Young Investigator Award
• Recognition Award for Outstanding Early Career Investigators
• Young Investigator Award
• Resident research award

Education

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• Nationwide Children's Hospital
• Indiana University School of Medicine

Hospital

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• Children's of Alabama
• University of Alabama Hospital

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